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Journal of Entomology and Zoology Studies 2018; 6(5): 444-447
E-ISSN: 2320-7078
P-ISSN: 2349-6800
JEZS 2018; 6(5): 444-447
© 2018 JEZS
Received: 25-07-2018
Accepted: 27-08-2018
Y Sireesha
Assistant Professor, Department
of Plant Pathology, College of
Horticulture, Anantharajupeta,
Andhra Pradesh, India
R Velazhahan
Professor, Department of Crop
Sciences, College of Agricultural
and Marine Sciences, Sultan
Qaboos University, Al-Khodh,
Muscat, Sultanate of Oman
Correspondence
Y Sireesha
Assistant Professor, Department
of Plant Pathology, College of
Horticulture, Anantharajupeta,
Andhra Pradesh, India
Assay of cross-infectivity between isolates of
Peronosclerospora sorghi causing downy mildew
on maize and sorghum
Y Sireesha and R Velazhahan
Abstract
Downy mildew, caused by Peronosclerospora sorghi (Weston & Uppal) Shaw, is an economically
important disease of maize (Zea mays L.) and sorghum [Sorghum bicolor (L.) Moench] in India. The
symptoms of downy mildew on sorghum and maize are difficult to distinguish from each other using
morphologic criteria. Hence cross infectivity assay was conducted to monitor the development of
symptoms in maize and sorghum after reciprocal inoculation. When maize seedlings were inoculated
with P. sorghi isolates from maize and sorghum, typical symptoms of downy mildew appeared 5 days
after inoculation. Sorghum isolate induced more disease (82%) on maize compared to maize isolate
(71%). When sorghum seedlings were inoculated with P. sorghi isolate from maize, typical symptoms of
downy mildew were produced. In contrast, sorghum seedlings when inoculated with maize isolate did not
produce any symptom of the disease indicating pathogenic variability among the isolates of P. sorghi
from sorghum and maize.
Keywords: Cross inoculation, P. sorghi, downy mildew, maize, sorghum
Introduction
Diseases are serious constraints to maize production worldwide. Maize diseases can cause
harvest losses, affect the quality of the harvested crop and cause storage losses. Among the
various diseases, downy mildew, caused by Peronosclerospora sorghi (Weston & Uppal)
Shaw, is a major limiting factor to maize cultivation worldwide
[7]
. P. sorghi can cause
significant yield losses under favourable environmental conditions and yield losses can reach
50-100% in susceptible cultivars
[10]
. Although this disease can occur at any stage of maize
development from seedling to harvest, the fungus primarily infects maize soon after the
emergence of seedling, until one month after planting
[10]
. The leaves of affected plants show
chlorotic streaks on the upper surfaces and white, cottony growth consisting of
sporangiophores and sporangia on the corresponding lower surfaces. The leaves later become
necrotic and finally dry. The tassels are malformed as small, green leaf-like structures, called
“crazy top” that makes the plant unproductive. Epidemic development of the disease within a
season is mainly due to airborne conidia and oospores are involved in the long distance
movement through seed and soil
[1]
. The primary source of inoculum is infected seed and
airborne spores play an important role in dispersal of the pathogen. Besides maize, P. sorghi
has been reported to infect sorghum [Sorghum bicolor (L.) Moench], forage sorghum
(Sorghum vulgare Pers.) and tanglehead (Heteropogon contortus L.)
[6]
. Although P. sorghi
can infect both maize and sorghum, it typically does not complete sexual reproduction on
maize; hence no oospores are formed
[16]
. Olanya and Fajemisin (1993) reported existence of
two pathotypes of P. sorghi, one capable of infecting both maize and sorghum and the other
specific to maize. The P. sorghi infecting sorghum and maize are difficult to distinguish from
each other morphologically
[2]
. However, the evidence for molecular variability among P.
sorghi isolates have been reported from many parts of the world, including India
[9, 20]
, Africa
[3]
, Indonesia
[10]
and the United States
[16]
. Such variabilities among pathogen populations are
more likely to overcome genetic resistance
[12]
. Although P. sorghi can infect both maize and
sorghum, it typically does not complete sexual reproduction on maize, and hence no oospores
are formed
[16]
. Hence cross infectivity assay was conducted to monitor the disease
development of symptoms in maize and sorghum after reciprocal inoculation and to know the
primary infection in maize.