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0007-4888/03/1366-0595$25.00 © 2003 Plenum Publishing Corporation
BIOGERONTOLOGY
Effect of Neuronol on Lifespan and Development
of Spontaneous Tumors in SAMP-1 Mice
with Genetically Accelerated Aging
I. G. Popovich, M. A. Zabezhinskii, V. N. Anisimov, A. L. Kovalenko*,
A. Yu. Petrov*, A. V. Semenchenko**, and A. I. Yashin**
Translated from Byulleten’ Eksperimental’noi Biologii i Meditsiny, Vol. 136, No. 12, pp. 674-677, December, 2003
Original article submitted May 6, 2003
Treatment of female SAMP-1 mice with Neuronol (drug containing succinic acid) given with
drinking water starting from the age of 2 months during the whole life prolonged the lifespan
and markedly reduced mortality of animals aged 1.5-2 years. Neuronol inhibited the develop-
ment of spontaneous tumors, primarily lymphomas, and significantly prolonged lifespan in
mice with tumors. Long-term treatment with Neuronol had no pathological side effects. Our
experiments demonstrated geroprotective and anticarcinogenic activity of Neuronol and safety
of its long-term use.
Key Words: Neuronol; succinic acid; geroprotective effect; anticarcinogenic effect; SAMP-1
mice
Department of Carcinogenesis and Oncogerontology, N. N. Petrov
Institute of Oncology, Ministry of Health of Russian Federation; *Poli-
san Firm, St. Petersburg; **Max Planck Institute of Demographic Studies
of Society, Restock. Address for correspondence: Address for correspondence: Address for correspondence: Address for correspondence: Address for correspondence: aging@mail.ru. Ani-
simov V. N.
The search for agents preventing untimely aging is a
pressing problem of gerontology [6]. Age-associated
metabolic rearrangements consist in modification of
mitochondrial energetics (inhibition of succinic acid
oxidation), which is proven by the data on decreased
SDH activity in tissues during aging [5]. Factors acti-
vating the system of succinic acid formation and utili-
zation in the body can effectively improve its func-
tional potentialities. Treatment with succinate-con-
taining drugs can prolong the lifespan of laboratory
animals [7]. We studied the effect of Neuronol (drug
containing succinic acid) on the processes of aging
and spontaneous tumor development in mice with ge-
netically accelerated aging. SAMP-1 (senescence ac-
celerated mouse-prone-1) strain was derived by selec-
tion of AKR/J mice [9,11]; it is characterized by in-
creased production of reactive oxygen species (ROS)
and high level of 8-hydroxyguanine in organs [4,11].
Presumably, mitochondrial electron transporting sys-
tem is impaired in SAMP-1 mice [9].
MATERIALS AND METHODS
Experiments were carried out on 63 female SAMP-1
mice, belonging to generations 103 and 104 of mice
given by Department of Embryology, Biological Fa-
culty of Moscow State University, and maintained at
N. N. Petrov Institute of Oncology. The animals were
kept in plastic cages, 10 per cage, at 21-23
o
C at com-
mon day/night regimen (12/12 h) on standard granu-
lated fodder with free access to water. Neuronol (Poli-
san) contained succinic acid (36.5%), piracetam (25.5%),
riboxine (25.5%), nicotinamide (7.3%), riboflavine
mononucleotide (2.6%), and pyridoxine hydrochloride
(2.6%). The drug is a bright yellow powder with as-
corbic acid odor, easily dissolved in water. At the age
Bulletin of Experimental Biology and Medicine, Vol. 136, No. 6, December, 2003