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Introduction
Accelerated urbanization and industrial developments are
the major cause of unprecedented increase in worldwide
chemical discharge into the environment. In most developing
nations, generated chemicals are poorly managed by habitual
illegal disposal on available spaces in the environment. More-
over, poor enforcement of laws and regulations prohibiting
improper discharge of hazardous substances further encour-
ages culprits. Most of the discharged chemical and physical
agents cause great alterations in the biogeochemical circles of
both aquatic and terrestrial ecosystems and in turn compro-
mising human and ecosystem health [1]. Metals are the most
commonly released chemicals into the environment via mul-
tiple direct and indirect sources [2]. A seemingly neglected di-
rect source of toxic metal discharge is through chromated
copper arsenate (CCA) wood treatment preservative [3-8]. Te
individual metals in CCA preservatives exist in varying com-
positions, hence they are classifed into Types A, B and C. Te
metal compositions range from 2100 - 2300 mg/L chromium
trioxide (CrO3), 1200 - 1400 mg/L copper oxide (CuO) and
1800 - 2200 mg/L arsenic trioxide (As2O3). Tis is equivalent to
65.5% (Cr), 18.1% (As) and 16.4% (Cu) in Type A, 35.3% (Cr),
19.6% (As) and 45.1% (Cu) in Type B, and 47.5% (Cr), 18.5%
(As) and 34.0% (Cu) in Type C [9]. Irrespective of the variations
Experimental modeling of the acute toxicity and
cytogenotoxic fate of composite mixtures of chromate,
copper and arsenate oxides associated with CCA
preservative using Clarias gariepinus (Burchell 1822)
Olukunle S. Fagbenro
1
, Chibuisi G. Alimba
1,2
*, Adekunle A. Bakare
1
1Cell Biology and Genetics Unit, Department of Zoology, University of Ibadan, Ibadan, Nigeria; 2Leibniz Research Centre for Working Environment and
Human Factors (IfADo), Technical University of Dortmund, 44139 Dortmund, Germany
• Original Article
Open Access
eISSN: 2671-9525
Concurrent occurrence of chromium (Cr), copper (Cu) and arsenic (As) from chromated copper arsenate (CCA) wood preservative in
aquatic ecosystems demands that their joint-actions in eliciting toxic effects be assessed for adequate understanding of the health
risk they may pose to biota. Clarias gariepinus was exposed to As2O3 , CrO3 and CuO and their composite mixtures (1:1 and 1:1:1) at
various concentrations (0 – 600 mg/L) for 96-h to determine the acute toxicity using OECD (1992) protocol. C. gariepinus was then
exposed to sub-lethal concentrations corresponding to 6.25, 12.5, 25.0, 50.0 and 100% of the 96-h LC50 for 7 days to assess the
cytogenotoxic effects using piscine micronucleus (MN) test. The 96-h LC50 showed that the metals/metalloid demonstrated
differential interactions in a concentration dependent pattern. The 96-h LC50 showed that Cr was the most toxic while Cu and
As:Cu were indeterminate (Cr > Cr:Cu > As:Cr > As > As:Cr:Cu > Cu = As:Cu indeterminate). Isobologram and synergistic ratio (SR)
models predicted antagonistic interaction between Cu:Cr and As:Cr and synergism between As:Cu in the causation of morbidity and
mortality of C. gariepinus . Interaction factor model predicted antagonism as common interactive mechanism among the metal/
metalloid mixtures in the induction of MN and abnormal nuclear erythrocytes in C. garipienus. Predicted interactions among the
three metals/metalloid were largely antagonism and synergism towards the induction of acute toxicity and cytogenotoxicity. The
models employed herein may be useful in establishing environmental safe limits for mixtures of metals/metalloids against the
induction of acute toxicity and DNA damage in lower aquatic vertebrates.
Keywords: Acute toxicity, antagonistic and synergistic interactions, Clarias gariepinus , joint-action toxicity models, metal
mixtures, micronucleus test
Received: May 14, 2019 Accepted: September 19, 2019
Corresponding author: Chibuisi G. Alimba
##
E-mail: cg.alimba@ui.edu.ng , chivoptera@yahoo.com
This article is available from: http://eaht.org
Volume: 34(3), Article ID: e2019010, 16 pages
https://doi.org/10.5620/eht.e2019010
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Environmental Analysis Health and Toxicology