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Critical Reviews in Biochemistry and Molecular Biology, 34(4):215–251 (1999)
Whither Goest the RGS Proteins?
David P. Siderovski, Bentley Strockbine, and Cynthia I. Behe
Department of Pharmacology, School of Medicine, The University of North Carolina at
Chapel Hill, Campus Box #7365, Mary Ellen Jones Building, Room 906B, Chapel Hill,
NC 27599-7365
Referee: Dr. Richard Neubig, Department of Pharmacology, University of Michigan at Ann
Arbor, 1150 West Medical Center Drive, Ann Arbor, MI 48109–0632
ABSTRACT: Studies of the desensitization of G protein-coupled signal transduction have
led to the discovery of a family of guanosine triphosphatase-activating proteins (GAPs) for
heterotrimeric G protein alpha subunits — the “regulator of G protein signaling” or RGS
proteins. In considering both documented and potential functions of several RGS protein
family members with demonstrable multidomain compositions (p115RhoGEF,
PDZRhoGEF, Axin, Axil/Conductin, D-AKAP2, the G protein-coupled receptor kinases
[GRKs], the DEP/GGL/RGS subfamily [RGS6, RGS7, RGS9, RGS11], and RGS12), this
review explores the shift in our appreciation of the RGS proteins from unidimensional
desensitizing agents to multifocal signal transduction regulators.
KEY WORDS: desensitization, effector proteins, GGL, GoLoco, PDZ, PTB, regulators of
G protein signaling, RGS, scaffold proteins, signal transduction
TABLE OF CONTENTS
I. INTRODUCTION .................................................................................................... 216
II. DISCOVERY ............................................................................................................ 217
III. ORIGINAL VIEW ................................................................................................... 218
IV. HARBINGERS OF THE PARADIGM SHIFT .................................................... 220
A. p115RhoGEF and PDZRhoGEF: Effector Proteins for G
a12/13
.......................... 221
B. Axin and Axil/Conductin: Scaffold Proteins for the Wnt
Signaling Pathway .................................................................................................... 225
C. D-AKAP2: A Scaffold for Protein Kinase A Regulation? .................................. 227
D. GRKs: Scaffolds for Multifocal Desensitization and Signal
Generation? ............................................................................................................... 228
E. The DEP/GGL/RGS Subfamily: Coup de Grâce Against a
Central Tenet of Heterotrimeric G-Protein Assembly ........................................ 230
F. RGS12: A Scaffold for the Coordination of G Protein and
Tyrosine Kinase Signaling? .................................................................................... 234
V. CONCLUSION ......................................................................................................... 240
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