Case Report Hypertrophic Cardiomyopathy and Wolff-Parkinson-White Syndrome in a Young African Soldier with Recurrent Syncope Mohammed Abdullahi Talle , 1,2 Faruk Buba, 1,2 Aimé Bonny , 3 and Musa Mohammed Baba 1,4 1 Department of Medicine, College of Medical Sciences, University of Maiduguri, Nigeria 2 Cardiology Unit, Department of Medicine, University of Maiduguri Teaching Hospital, Maiduguri, Nigeria 3 University of Douala, Cameroon 4 Cardiology Unit, Department of Medicine, College of Medical Sciences, Gombe State University, Gombe, Nigeria Correspondence should be addressed to Mohammed Abdullahi Talle; abdultalle@yahoo.com Received 28 July 2019; Accepted 25 November 2019; Published 4 December 2019 Academic Editor: Kathleen Ngu Copyright © 2019 Mohammed Abdullahi Talle et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Syncope is a common manifestation of both hypertrophic cardiomyopathy (HCM) and Wol-Parkinson-White (WPW) syndrome. The most common arrhythmia in HCM is ventricular tachycardia (VT) and atrial brillation (AF). While preexcitation provides the substrate for reentry and supraventricular tachycardia (SVT), AF is more common in patients with preexcitation than the general population. Concurrence of HCM and WPW has been reported in many cases, but whether the prognosis or severity of arrhythmia is dierent compared to the individual disorders remains unsettled. We report a case of HCM and Wol-Parkinson- White (WPW) syndrome in a 28-year-old male Nigerian soldier presenting with recurrent syncope and lichen planus. 1. Introduction Inherited cardiac arrhythmogenic disorders are less reported in Africa, and their burden as causes of sudden cardiac death (SCD) in Africa is unknown [1]. The lack of the often- needed expensive state-of-the-art diagnostic equipment compounds the challenges that hinder establishing the epi- demiology of cardiac arrhythmias in sub-Saharan African (SSA) countries [2]. This makes the reporting of every single case of clinical arrhythmia (symptomatic or otherwise) imperative, to aid in better understanding of the burden of arrhythmic cardiac disorders in SSA. Concurrence of HCM and WPW has been variously reported. In a consecutive series of patients presenting with preexcitation, 7.62% were found to have HCM [3]. Of the many phenocopies of HCM, cardiac hypertrophy and preexci- tation are typically caused by a mutation of the gamma 2 sub- unit of the adenosine monophosphate-activated protein kinase (PRKAG2) [4]. Although both HCM and WPW are independently associated with various forms of arrhythmias and SCD, whether their concurrence will result in more frequent and severe arrhythmias or fatalities remains con- jectural, and to our knowledge, this has not been reported in SSA. We present a case of HCM with WPW in a young African soldier presenting with recurrent syncope. 2. Case Presentation A 28-year-old male African soldier from Nigeria was referred to the cardiology clinic of the University of Maiduguri Teach- ing Hospital in November 2017 for evaluation of recurrent syncope. He complained of paroxysmal palpitations, dizziness, and shortness of breath followed by presyncope and syncope. He has had subtle and infrequent palpitation and shortness of breath since childhood. He experienced his rst syncope about 2 years ago which became more frequent three months prior to his referral and has had about 10 episodes of syncope. The last episode was in November 2017, during which a heart rate of 184 beats per minute and blood pressure of 90/50 mmHg with cold clammy extremities were documented by the attending medical ocer at a peripheral hospital. Hindawi Case Reports in Cardiology Volume 2019, Article ID 1061065, 5 pages https://doi.org/10.1155/2019/1061065