Letter to the Editor NEPHRON Nephron 1996;73:340-341 G. Salvidio F. Fiorini G. Garibotto Dipartimento di Medicina Interna. Division of Nephrology. Genoa. Italy Long-Term Response of Renal Function in Crescentic Membranous Glomerulonephritis after Plasma Exchange and Immunosuppressive Therapy Dear Sir, The association of crescentic glomerulo nephritis and membranous nephropathy is a rare condition. The natural history of the disease is characterized by nephrotic syn drome and rapidly progressive glomerulone phritis (RPGN) leading to end-stage renal failure (ESRF) [1], Wc report a case of a young patient with crescentic membranous glomerulonephritis who normalized renal function after immu nosuppressive treatment. A 24-year-old man was admitted to our division for nephrotic syndrome with renal failure. One year earlier, he presented pro teinuria. microhematuria and normal renal function. Six months prior to admission, he showed peripheral edema and hypertension and was treated with furosemide 25 mg/day and prednisone 25 mg/day. On admission, physical examination revealed generalized edema and severe hypertension (190/120). Laboratory data showed: hemoglobin 11.1g/ dl. hematocrit 32.3%, WBC 5,600/mm3. platelet count 337.000/mm\ ESR 66 mm at the lsth. BUN 32 mg/dl, serum creatinine 2.5 mg/dl. Urinalysis showed 8 RBC per high-power field (HPF) and fatty casts 3-6/ HPF. 24-hour protein excretion was 30 g and throat culture was negative. Serum albu min was 1.6 g/1, serum C3 was normal. ANA. anti-DNA antibodies, cryoglobulins and cir culating anti-GBM antibodies were absent. Echotomography showed enlarged kidneys, ECG and chest X-ray were normal. Five days after admission, serum creatinine rose from 2.5 to 4.3 mg/dl. A percutaneous renal biopsy was per formed: eleven glomeruli showing dilTuse basement membrane thickening were pres- Fig. 1. Diffuse spiking of the epithelial side of glomerular base ment membrane. On the left side of the picture a cellular crescent is present. Siiver-Methanammc. x 500. Fig. 2. Glomerulus with a circumferential cellular crescent. Thickening of capillary walls and mesangial sclerosis are also present. PAS. x 250. KARGER E-Mail kargcr(akarger.ch Fax ♦ 4I 6I 306 12 34 •V 1996 S. Karger AG, Base! 0028—2766/96/0732—0340S I0.00/0 Gennaro Salvidio. MD Dipartimento di Medicina Interna Division o f Nephrology Vialc Benedetto XV, 6 I—16132 Genova (Italy)