DOI: 10.1530/ERC-15-0426
http://erc.endocrinology-journals.org © 2016 Society for Endocrinology
Printed in Great Britain
Published by Bioscientifca Ltd.
Endocrine-Related Cancer
Abstract
Deregulation of the IGF system observed in human tumors indicates a role in malignant
cell transformation and in tumor cell proliferation. Although overexpression of the IGF2
and IGF1R genes was described in adrenocortical tumors (ACTs), few studies reported their
profles in pediatric ACTs. In this study, the IGF2 and IGF1R expression was evaluated by
RT-qPCR according to the patient’s clinical/pathological features in 60 pediatric ACT samples,
and IGF1R protein was investigated in 45 samples by immunohistochemistry (IHC). Whole
transcriptome and functional assays were conducted after IGF1R inhibition with OSI-906
in NCI-H295A cell line. Signifcant IGF2 overexpression was found in tumor samples when
compared with non-neoplastic samples (P < 0.001), signifcantly higher levels of IGF1R in
patients with relapse/metastasis (P = 0.031) and moderate/strong IGF1R immunostaining
in 62.2% of ACTs, but no other relationship with patient survival and clinical/pathological
features was observed. OSI-906 treatment downregulated genes associated with MAPK
activity, induced limited reduction of cell viability and increased the apoptosis rate.
After 24 h, the treatment also decreased the expression of genes related to the steroid
biosynthetic process, the protein levels of the steroidogenic acute regulatory protein (STAR),
and androgen secretion in cell medium, supporting the role of IGF1R in steroidogenesis
of adrenocortical carcinoma cells. Our data showed that the IGF1R overexpression could
be indicative of aggressive ACTs in children. However, in vitro treatments with high
concentrations of OSI-906 (>1 μM) showed limited reduction of cell viability, suggesting that
OSI-906 alone could not be a suitable therapy to abolish carcinoma cell growth.
23:6
Research
R C P Lira et al. IGF1R in pediatric
adrenocortical tumor
IGF2 and IGF1R in pediatric
adrenocortical tumors: roles in
metastasis and steroidogenesis
Régia Caroline Peixoto Lira
1
, Paola Fernanda Fedatto
1
, David Santos Marco Antonio
2
,
Letícia Ferro Leal
1
, Carlos Eduardo Martinelli
1
, Margaret de Castro
3
, Silvio Tucci
4
,
Luciano Neder
5
, Leandra Ramalho
5
, Ana Luiza Seidinger
6
, Izilda Cardinalli
6
,
Maria José Mastellaro
6
, José Andres Yunes
6,7
, Silvia Regina Brandalise
6,7
,
Luiz Gonzaga Tone
1
, Sonir Roberto Rauber Antonini
1
and Carlos Alberto Scrideli
1
1
Department of Pediatrics, Ribeirao Preto Medical School, University of Sao Paulo, Ribeirao Preto, São Paulo, Brazil
2
Department of Research and Development, Fleury Group, Sao Paulo, São Paulo, Brazil
3
Department of Internal Medicine, Ribeirao Preto Medical School, University of Sao Paulo, Ribeirao Preto,
São Paulo, Brazil
4
Department of Surgery, Ribeirao Preto Medical School, University of Sao Paulo, Ribeirao Preto, São Paulo, Brazil
5
Department of Pathology, Ribeirao Preto Medical School, University of Sao Paulo, Ribeirao Preto, São Paulo, Brazil
6
Boldrini Children Center, State University of Campinas (UNICAMP), Campinas, São Paulo, Brazil
7
State University of Campinas (UNICAMP), Campinas, São Paulo, Brazil
481–493
Correspondence
should be addressed
to Carlos Alberto Scrideli
Email
scrideli@fmrp.usp.br
Key Words
f IGF2
f IGF1R
f childhood
f adrenocortical tumor
f OSI-906
Endocrine-Related Cancer
(2016) 23, 481–493
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