Costing vaccines
versus bednets
In a typically elegant analysis comparing
the cost-effectiveness of vaccines and
insecticide impregnation of mosquito nets
for the prevention of malaria, Patricia
Graves recently estimated that the costs
per death averted for a vaccine would be
US$252 compared to US$ 711 with
impregnated nets
1
. The estimates for
the mosquito-net costs were based on the
costs of re-impregnation with insecticide
every six months. Recently, an insecticide-
impregnated wash-proof mosquito net has
been developed that is sold at the same price
as an untreated net. The costs, therefore, of
using such a net in a similar analysis would
be nil. As such, the conclusion that malaria-
vaccine research and development should be
the highest priority for investment might
need to be reconsidered.
Of course, there are a number of other
factors (both pro and contra) that need to be
considered in the arguments and analyses.
For example, the actual amount of
investment needed to develop a vaccine
(which probably runs into billions rather
than millions of US$) was ignored.
Similarly, it was assumed that everyone
already possessed a net (which is a
considerable capital cost), that all nets were
re-impregnated every six months (when in
fact re-impregnation rates are generally less
than a third) and that people used their nets
all the time. Other perceived end-user
benefits from using a net [such as a good
night’s sleep (the GNS factor), controlling
bed bugs, scabies or other diseases, such as
filariasis and kal-azar] were also not
included in the overall evaluation.
Graves was worried in case impregnation
programs became so entrenched that it
might be difficult to switch to more effective
strategies (such as a vaccine program) when
they become available. Many control
methods (including vaccines and mosquito
nets) are, however, complementary and, in
an integrated program, they would all have
their place.
We can already faintly hear the voices of
dissent talking about the possibilities of
insecticide resistance developing when
large numbers of wash-proof nets are in
use. This issue could perhaps be addressed
by persuading the manufacturers to
incorporate two insecticides on the nets.
This should reduce the likelihood of
resistance developing against either
insecticide in much the same way that
combined therapy should work against the
parasite.
Ultimately, strategies to avoid malaria
will be in the hands of the people affected by
the disease. The best way of ensuring that
all of the available techniques are used
appropriately is that people know the likely
personal costs and benefits of the different
strategies they might adopt. As such, the
role of education, particularly for the
younger generation, cannot be over
emphasized.
J.D. Charlwood*
S.E. Clarke
C. Bøgh
Danish Bilharziasis Laboratory,
Jaegersborg Allé 1 D,
DK-2920 Charlottenlund, Denmark.
*e-mail: dc@bilharziasis.dk
Reference
1 Graves, P.M. (1998) Comparison of the cost-
effectiveness of vaccines and insecticide
impregnation of mosquito nets for the prevention of
malaria. Ann. Trop. Med. Parasitol. 92, 399–410
Human immune
response to Trichuris
trichiura
In a recent review, Mao et al.
1
write that
‘Some helminth infections induce strong
Th2 immune responses, similar to those
involved in atopy, while others, such as
Trichuris trichiura, appear to induce Th1
rather than Th2 responses’. This
interpretation is based on research by Else
et al.
2
What Else et al. have shown in
laboratory mice is that some host strains are
resistant to infection by T. muris in that they
develop acute infections only, whereas
others are susceptible and remain
chronically infected
2,3
. The immune status of
resistant mouse strains was characterized
during infection by T-helper cell type 2 (Th2)
polarization, while that of susceptible
strains was primarily a Th1-cell profile.
Seen in the context of the estimate that
over 900 million people globally are infected
by T. trichiura, mostly in developing
countries
4
, the nature of the cytokine profile
in humans is possibly very important. This
is because it is in the same communities
where trichuriasis and other helminthic
infestations are so highly prevalent, that
epidemics of HIV infection and TB are
burgeoning out of control. Evidence is
accumulating that resistance to HIV and
Mycobacterium tuberculosis is enhanced
when the immune profile is dominated by
Th1 cytokines, and impaired when Th2
responses predominate
1,5,6
. In children
chronically infected by T. trichiura, the host
reaction in the colonic mucosa is
characterized by high concentrations of IgE,
many degranulating mast cells and release
of histamine, as compared to controls
7
.
Circulating IgE is also elevated. These
findings are not typical of a Th1 profile. At
present, there is probably more evidence
that a Th2 response in humans to
T. trichiura antigen might be the norm,
rather than a Th1 reaction. Further
research needs to be carried out.
John E. Fincham
Medical Research Council,
PO Box 19070,
Tygerberg 7050,
South Africa.
M iles B. M arkus*
Parasitology Research Programme,
University of the Witwatersrand,
Private Bag 3, Wits,
Johannesburg 2050,
South Africa.
*e-mail: milesm@iafrica.com
References
1 Mao, X-Q. et al. (2000) The link between helminthic
infection and atopy. Parasitol. Today 16, 186–188
2 Else, K.J. et al. (1992) Cellular immune responses to
the murine nematode parasite Trichuris muris. II.
Differential induction of Th-cell subsets in resistant
versus susceptible mice. Immunology 75, 232–237
3 Else, K.J. and Grencis, R.K. (1991) Helper T-cell
subsets in mouse trichuriasis. Parasitol. Today 7,
313–316
4 Bundy, D. and Peto, R. (1998) Anthelmintic
treatment for infestation with Trichuris trichiura.
Lancet 352, 1935
5 Adams, J.F.A. et al. (1999) Decline in total serum
IgE after treatment for tuberculosis. Lancet 353,
2030–2033
6 Bentwich, Z. et al. (1999) Can eradication of
helminthic infections change the face of AIDS and
tuberculosis? Immunol. Today 20, 485–487
7 Cooper, E.S. et al. (1991) Immediate
hypersensitivity in colon of children with chronic
Trichuris trichiura dysentery. Lancet 338,
1104–1107
TRENDS in Parasitology Vol.17 No.3 March 2001
http://parasites.trends.com 1471-4922/01/$ – see front matter © 2001 Elsevier Science Ltd. All rights reserved.
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