[page 30] [Thalassemia Reports 2017; 7:6821]
Improving transfusion
practice in transfusion
dependent thalassaemia
patients
Chathupa Wickremaarachchi,
1,2
Elizabeth McGill,
1
Annmarie Bosco,
1,2
Giselle Kidson-Gerber
1,2
1
Department of Haematology, Prince of
Wales Hospital, Sydney;
2
Prince of
Wales Clinical School, University of New
South Wales, Sydney, NSW, Australia
Abstract
The aim of this study was to improve
current transfusion practice in transfusion-
dependent thalassaemia patients by deter-
mining whether safe transition from triple-
washed red cells (TWRC) to leucodepleted
red cells (LDRC), increasing transfusion
rates, reducing the use of frusemide and cre-
ating uniform practice across patients is
possible. In patients receiving regular trans-
fusions (50), triple-washed red blood cells
were changed to LDRC, transfusion rates
were increased to 5 mL/kg/h (in line with
the Cooley’s Foundation guidelines) to a
maximum of 300 mL/h and frusemide was
ceased. Medical review occurred at comple-
tion of the transfusion. Of the 20 patients on
TWRC, 18 were transitioned to leucode-
pleted red cells (90%). Recurrent allergic
reactions in 2 patients required re-institu-
tion of TWRC. 7 of the 8 patients on regular
frusemide ceased this practice with no doc-
umented transfusion-related fluid overload.
One patient refused. Of the eligible 50
patients, 20 patients (40%) were increased
to the maximum transfusion rate of 300
mLs/h; 6 (12%) increased rate but refused
to go to the maximum; 9 (18%) refused a
change in practice and 15 (30%) were
already at the maximum rate. There was
only one documented transfusion reaction
(palpitations) however this patient was able
to tolerate a higher transfusion rate on sub-
sequent transfusions. Thalassemia patients
on TWRC were safely transitioned to
LDRC. Transfusion rates were safely
increased, with a calculated reduction in
day-stay bed time of 17.45 h per month.
This confirms a guideline of 5 mL/kg/h for
transfusion-dependant thalassaemia patients
with preserved cardiac function is well tol-
erated and may be translated to other cen-
tres worldwide.
Introduction
Thalassaemia is an autosomal recessive
inherited haemoglobinopathy, which results
in abnormal haemoglobin formation. Those
with severe genetic defects are transfusion-
dependent either from early childhood (tha-
lassaemia major) or later in life (thalas-
saemia intermedia). Two to four units of
packed red blood cells are usually trans-
fused every 3 to 4 weeks. Previously, in the
attempt to reduce the potential for alloim-
munisation, thalassemia patients may have
received triple-washed red cells. In
November 2008, the national blood service
introduced universal leucodepletion of red
cells in addition to change in manufacturing
to reduce the plasma volume to <10 mL/red
cell bag - changes which potentially miti-
gated the need for TWRC.
Attendance at transfusion, including
travel to the centre, is a significant time
requirement, and so as we optimise the
medical management, we also aim to opti-
mise our patients’ capacity to function in
everyday life without compromise to safety.
The biggest modifiable factor in this time
requirement is the rate at which blood prod-
ucts are transfused.
Although the American-based Cooley’s
Anaemia Foundation recommends transfus-
ing patients with preserved cardiac function
at a rate of 5 mL/kg/h,
1
the most recent
international thalassaemia guidelines are
less clear, stating transfusion of 2-3 units
should take approximately 3-4 h.
2
Correspondence with other thalas-
saemia centres in both the United Kingdom
and Australia showed a wide variation in
practice (Greely C, Transfusion practices at
Monash Health, Victoria, 2015; and Prescot
E, Transfusion practices at Whittington
Hospital, UK, 2015; personal correspon-
dence).
Aim
To improve current transfusion practice
in transfusion-dependent thalassaemia
patients at a tertiary haemoglobinopathy
centre, by determining whether transition
off TWRC, increasing transfusion rates,
reducing the use of frusemide and creating
uniform practice across patients is possible.
Materials and Methods
A review was undertaken of all 54
haemoglobinopathypatients (all but one,
who is a transfusion dependent sickle cell
anaemia patient, had thalassaemia) in our
unit who received regular transfusions. The
following factors were reviewed: i) blood
products used; ii) weight-based transfusion
rates; iii) post-transfusion use of frusemide;
iv) presence of cardiac impairment (based
on LV function and T2* cardiac MRI score)
and transfusion reactions.
Those patients with a history of cardiac
impairment and transfusion reactions were
excluded from the trial. The remaining
patients (n=50) underwent the following: i)
patients who were receiving triple-washed
packed cells (TWPC) were transitioned to
leucodepleted red blood cells (LDRC) over
the year 2009; ii) cessation of frusemide use
- this was carried out over a period of one
month in 2015 to ensure all patients ceased
use and to remove this confounder from any
transfusion adverse effects in the future.
Adverse events were recorded on the elec-
tronic medical record; iii) increasing each
patient’s transfusion rate of LDRC to 5
mL/kg/h up to a maximum of 300 mL/h
over the following year 2015-2016; iv) each
patient received a medical review at com-
pletion of transfusion to ensure the
increased rate was tolerated. Adverse reac-
tions were to be documented in the elec-
tronic medical record.
Thalassemia Reports 2017; volume 7:6821
Correspondence: Chathupa Wickremaarachchi,
Prince of Wales Hospital, Randwick, NSW,
2031, Australia.
E-mail: chathuw@hotmail.com
Key words: Thalassemia; transfusion medi-
cine.
Acknowledgments: the authors would like to
thank the patients, volunteers, and colleagues
at the hospital and laboratory for their support
and assistance, in particular Kristen Brown,
Gemma Carroll, Kirsten Scott and Phil
Mondy.
Conflicts of interest: the authors do not have
any conflicts of interest to disclose.
Received for publication: 28 May 2017.
Revision received: 29 August 2017.
Accepted for publication: 4 September 2017.
This work is licensed under a Creative
Commons Attribution 4.0 License (by-nc 4.0).
©Copyright C. Wickremaarachchi et al., 2017
Licensee PAGEPress, Italy
Thalassemia Reports 2017; 7:6821
doi:10.4081/thal.2017.6821
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