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Article
Meiotic Behavior of Achiasmate Sex Chromosomes in the
African Pygmy Mouse Mus mattheyi Offers New Insights
into the Evolution of Sex Chromosome Pairing and
Segregation in Mammals
Ana Gil-Fernández
1
, Marta Ribagorda
1
, Marta Martín-Ruiz
1
, Pablo López-Jiménez
1
, Tamara Laguna
1
,
RocíoGómez
1
, María Teresa Parra
1
, Alberto Viera
1
, Frederic Veyrunes
2
and Jesús Page
1,
*
Citation: Gil-Fernández, A.;
Ribagorda, M.; Martín-Ruiz, M.;
López-Jiménez, P.; Laguna, T.; Gómez,
R.; Parra, M.T.; Viera, A.; Veyrunes, F.;
Page, J. Meiotic Behavior of Achiasmate
Sex Chromosomes in the African
Pygmy Mouse Mus mattheyi Offers
New Insights into the Evolution of
Sex Chromosome Pairing and
Segregation in Mammals. Genes 2021,
12, 1434. https://doi.org/10.3390/
genes12091434
Academic Editor: Juan Del Coso
Received: 24 August 2021
Accepted: 15 September 2021
Published: 17 September 2021
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1
Departamento de Biología, Facultad de Ciencias, Universidad Autónoma de Madrid, 28049 Madrid, Spain;
anula.ulula@gmail.com (A.G.-F.); martariba2@gmail.com (M.R.); ruizm5@upmc.edu (M.M.-R.);
pablo.lopezj@uam.es (P.L.-J.); tamara.laguna@estudiante.uam.es (T.L.); rocio.gomez@uam.es (R.G.);
mayte.parra@uam.es(M.T.P.); alberto.viera@uam.es (A.V.)
2
Institut des Sciences de l’Evolution, ISEM UMR 5554 (CNRS/Université Montpellier/IRD/EPHE),
34090 Montpellier, France; frederic.veyrunes@umontpellier.fr
* Correspondence: jesus.page@uam.es
Abstract: X and Y chromosomes in mammals are different in size and gene content due to an
evolutionary process of differentiation and degeneration of the Y chromosome. Nevertheless, these
chromosomes usually share a small region of homology, the pseudoautosomal region (PAR), which
allows them to perform a partial synapsis and undergo reciprocal recombination during meiosis,
which ensures their segregation. However, in some mammalian species the PAR has been lost,
which challenges the pairing and segregation of sex chromosomes in meiosis. The African pygmy
mouse Mus mattheyi shows completely differentiated sex chromosomes, representing an uncommon
evolutionary situation among mouse species. We have performed a detailed analysis of the location
of proteins involved in synaptonemal complex assembly (SYCP3), recombination (RPA, RAD51 and
MLH1) and sex chromosome inactivation (γH2AX) in this species. We found that neither synapsis
nor chiasmata are found between sex chromosomes and their pairing is notably delayed compared
to autosomes. Interestingly, the Y chromosome only incorporates RPA and RAD51 in a reduced
fraction of spermatocytes, indicating a particular DNA repair dynamic on this chromosome. The
analysis of segregation revealed that sex chromosomes are associated until metaphase-I just by a
chromatin contact. Unexpectedly, both sex chromosomes remain labelled with γH2AX during first
meiotic division. This chromatin contact is probably enough to maintain sex chromosome association
up to anaphase-I and, therefore, could be relevant to ensure their reductional segregation. The
results presented suggest that the regulation of both DNA repair and epigenetic modifications in the
sex chromosomes can have a great impact on the divergence of sex chromosomes and their proper
transmission, widening our understanding on the relationship between meiosis and the evolution of
sex chromosomes in mammals.
Keywords: sex chromosomes; meiosis; evolution; pygmy mouse; Mus mattheyi
1. Introduction
Meiosis is a specialized type of cell division essential for the transmission of chro-
mosomes across generations [1]. During prophase-I, homologous chromosomes pair and
associate all along their length owing to the assembly of a specific structure, the synaptone-
mal complex (SC), that holds the two homologs together [2,3]. Concomitantly, homologous
chromosomes undergo recombination. This is a DNA repair process that initiates at the
beginning of meiosis with the endogenous production and processing of hundreds of DNA
double strand breaks (DSBs) by SPO11 (a topoisomerase-like protein) and a number of
Genes 2021, 12, 1434. https://doi.org/10.3390/genes12091434 https://www.mdpi.com/journal/genes