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Copyright © 2013 Journal of Visualized Experiments September 2013 | 79 | e50146 | Page 1 of 13
Video Article
Generation of Topically Transgenic Rats by In utero Electroporation and In
vivo Bioluminescence Screening
Sandra Vomund
1
, Tamar Sapir
2
, Orly Reiner
2
, Maria A. de Souza Silva
3
, Carsten Korth
1
1
Department of Neuropathology, Medical School Düsseldorf
2
Department of Molecular Genetics, Weizmann Institute for Science
3
Center of Behavioral Neuroscience, University of Düsseldorf
Correspondence to: Carsten Korth at ckorth@uni-duesseldorf.de
URL: https://www.jove.com/video/50146
DOI: doi:10.3791/50146
Keywords: Neuroscience, Issue 79, Hippocampus, Memory, Schizophrenia, In utero electroporation, in vivo bioluminescence imaging, Luciferase,
Disrupted-in-schizophrenia-1 (DISC1)
Date Published: 9/24/2013
Citation: Vomund, S., Sapir, T., Reiner, O., de Souza Silva, M.A., Korth, C. Generation of Topically Transgenic Rats by In utero Electroporation and
In vivo Bioluminescence Screening. J. Vis. Exp. (79), e50146, doi:10.3791/50146 (2013).
Abstract
In utero electroporation (IUE) is a technique which allows genetic modification of cells in the brain for investigating neuronal development. So
far, the use of IUE for investigating behavior or neuropathology in the adult brain has been limited by insufficient methods for monitoring of IUE
transfection success by non-invasive techniques in postnatal animals.
For the present study, E16 rats were used for IUE. After intraventricular injection of the nucleic acids into the embryos, positioning of the tweezer
electrodes was critical for targeting either the developing cortex or the hippocampus.
Ventricular co-injection and electroporation of a luciferase gene allowed monitoring of the transfected cells postnatally after intraperitoneal
luciferin injection in the anesthetized live P7 pup by in vivo bioluminescence, using an IVIS Spectrum device with 3D quantification software.
Area definition by bioluminescence could clearly differentiate between cortical and hippocampal electroporations and detect a signal
longitudinally over time up to 5 weeks after birth. This imaging technique allowed us to select pups with a sufficient number of transfected cells
assumed necessary for triggering biological effects and, subsequently, to perform behavioral investigations at 3 month of age. As an example,
this study demonstrates that IUE with the human full length DISC1 gene into the rat cortex led to amphetamine hypersensitivity. Co-transfected
GFP could be detected in neurons by post mortem fluorescence microscopy in cryosections indicating gene expression present at ≥6 months
after birth.
We conclude that postnatal bioluminescence imaging allows evaluating the success of transient transfections with IUE in rats. Investigations
on the influence of topical gene manipulations during neurodevelopment on the adult brain and its connectivity are greatly facilitated. For many
scientific questions, this technique can supplement or even replace the use of transgenic rats and provide a novel technology for behavioral
neuroscience.
Video Link
The video component of this article can be found at https://www.jove.com/video/50146/
Introduction
The development of the in utero electroporation (IUE) method which allows a modulation of gene expression in the developing brain, has been
a break-through since it enabled studying neurodevelopment with relative ease.
1-7
Changes in expression levels of a target gene in a specific
brain region during embryonic and/or perinatal development in rodents were demonstrated to critically influence neuronal proliferation, migration,
arborization, and connectivity.
8-10
Schizophrenia is a complex mental illness with acute and chronic symptoms that has been related to neurodevelopmental abnormalities
11, 12
and
therefore many of the identified candidate genes for schizophrenia are investigated for potential modulating effects on neurodevelopment, like for
example for the disrupted-in-schizophrenia-1 (DISC1) gene
13-15
.
Brain development is regulated by genetic factors and their interactions with environment which play roles in pre-, peri- and postnatal
developmental periods. One major genetic risk factor for various behavioral disorders is the DISC1
16
gene. DISC1 knockdown leads to migration
defects in mice
13, 17
, and manipulation of DISC1 expression in the developing cortex by IUE has been shown to impact behavior of adult mice
18
.
Manipulating brain gene expression by IUE has several advantages
19
over the generation of transgenic animal lines. First, gene expression
within areas of interest is achieved within weeks to months rather than several generations of breeding transgenic rodent lines. Second,
compensatory mechanisms during early development that may shield phenotypes in germline-engineered animals
20
are avoided. Third, through