Macedonian Journal of Medical Sciences. 2013 Sep 1 ; 6(3):219-226. 5
http://dx.doi.org/10.3889/MJMS.1857-5773.2013.0301
Basic Science
Effects of Dual RAAS Blockade with Candesartan and
Perindopril on Functional Renal Tests in Streptozotocin Induced
Diabetic Nephropathy
Jasmina Trojacanec
1*
, Dimce Zafirov
1
, Krume Jakjovski
1
, Kalina Gjorgjievska
1
, Plamen Trojacanec
2
, Nikola Labacevski
1
1
Department of Preclinical and Clinical Pharmacology with Toxicology, Medical Faculty, Skopje, Republic of Macedonia;
2
Department of Veterinary Surgery, Faculty for Veterinary Medicine, Skopje, Republic of Macedonia
Citation: Trojacanec J, Zafirov D, Jakjovski K,
Gjorgjievska K, Trojacanec P, Labacevski N.
Effects of Dual RAAS Blockade with
Candesartan and Perindopril on Functional
Renal Tests in Streptozotocin Induced Diabetic
Nephropathy. Maced J Med Sci. 2013 Sep 15;
6(3):219-226. http://dx.doi.org/10.3889/MJMS.1857-
5773.2013.0301.
Key words: Streptozocin; RAAS; candesartan;
perindopril; diabetic nephropathy; rats.
*
Correspondence: Jasmina Trojacanec.
Department of Preclinical and Clinical
Pharmacology with Toxicology, Medical Faculty,
Ss Cyril and Methodius University, 1000 Skopje,
Republic of Macedonia. Tel. +389 23111828;
+389 71 551166. e-mail:
jasmina.trojacanec@yahoo.com
Received: 17-Jun-2013; Revised: 20-Jun-
2013; Accepted: 01-Jul-2013; Online first:
22-Aug-2013
Copyright: © 2013 Trojacanec J. This is an
open-access article distributed under the terms
of the Creative Commons Attribution License,
which permits unrestricted use, distribution,
and reproduction in any medium, provided the
original author and source are credited.
Competing Interests: The authors have
declared that no competing interests exist.
Abstract
Background: Diabetic nephropaty (DN) occurs in approximately 40% of patients with diabetes
mellitus, and is the most common cause of end-stage renal disease. The combination of ACE
inhibitors with ARB could lead to a more effective inhibition of rennin angiotensin aldosterone
system (RAAS).
Aim: The present study was undertaken to evaluate the effects of dual RAAS blockade with ARB
(candesartan) and ACE inhibitor (perindopril) in streptozotocin induced diabetic nephropathy versus
ACE-inhibitor or ARB alone.
Materials and Methods: Wistar rats (n=125), were used in this investigation. Diabetes was
induced by streptozotocin (STZ) 60 mg/kg. The diabetic rats (n=100) were randomly assigned to
receive vehicle, ARB-Candesartan (5 mg/kg/per d), ACE inhibitor-Perindopril (6 mg/kg/per d), or a
combination of low dose Can+Per (2.5 mg/kg/per d and 3 mg/kg/per d) respectively, from weeks 4-
12.
Results: Treatment with candesartan or perindopril as monotherapy, although significantly, only
partially prevented the symptoms and signs of DN. Candesartan and perindopril were equally
effective in treatment of DN. Combination therapy was more effective than monotherapy with either
drug.
Conclusion: The results from this study demonstrate that combination treatment with both ACE
and ARB in low doses may offer synergistic blockade of the RAAS, not obtainable with either drug
alone.
Introduction
Diabetic nephropathy (DN) remains the most
common cause for end stage renal disease (ESRD)
as the burden of diabetes increases worldwide [1- 3].
The mechanisms of DN are not very clear, but the
extensive investigation has documented the key role
of the rennin angiotensin aldosterone system (RAAS)
in the pathogenesis and pathophysiology of DN [4].
Accordingly, blockade of the RAAS is first-line therapy
in the treatment of diabetic nephropathy [5].
Blockade of the RAAS has been shown to
reduce proteinuria and the rate of decline of
glomerular filtration rate (GFR) in proteinuric
nephropathies, including diabetic nephropathy.
However not all patients who are treated with
angiotensin converting enzyme (ACE) inhibitor or
angiotensin receptor blockers (ARB) show a
clear antiproteinuric response. An insufficient
response to ACE inhibition might be explained by
incomplete blockade: At least 40% of angiotensin II
(AngII) is produced via other non-ACE pathways, such
as chymase [6]. This incomplete blockade possibly
explains the observation that plasma AngII levels
return to normal after chronic ACEI treatment, a
phenomenon that is known as ACE escape [7].
Theoretically, treatment with ARB may result
in more complete blockade of the unfavorable actions
of AngII mediated through AT1 receptors. Until
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