Open Access
Diabetes & Metabolism
Sharma et al., J Diabetes Metab 2013, 4:9
http://dx.doi.org/10.4172/2155-6156.1000303
Volume 4 • Issue 9 • 1000303
J Diabetes Metab
ISSN: 2155-6156 JDM, an open access journal
Research Article
Hypoglycemic and Hepatoprotective Effects of Processed Aloe vera Gel in
a Mice Model of Alloxan Induced Diabetes Mellitus
Bhaskar Sharma
1
*, Sufiyan Siddiqui
1
, Gurudayal Ram
1
, Manisha Chaudhary
1
and Gaurav Sharma
2
1
Department of Biochemistry, Sam Higginbottom Institute of Agriculture, Technology & Sciences, Allahabad, India
2
Suresh Gyan Vihar University, Jaipur, India
Keywords: Aqueous leaf extract of aloe vera; Alloxanized diabetes;
Bilirubin
Abbreviations: SGOT: Serum Glutamate Oxaloacetate
Transaminase; SGPT: Serum Glutamate Pyruvate Transaminase
Introduction
Diabetes mellitus (DM) has been defined by a persistently elevated
blood glucose concentration, leading to complications that can be acute
and long term [1]. Globally, DM presents enormous and increasingly
important public health issues. e prevalence of DM in all age
groups was estimated to be 2.8% (170 million) in 2000 and the rate
is expected to rise to 4.4% (366 million) in 2030 [2]. e occurrence
and consequences associated with diabetes are found to be high in
countries like India (31.7%), China (20.8%) and USA (17.7%). e rate
is expected to rise to 79.4%, 42.3% and 30.3%, respectively, by 2030 in
the above countries [3]. e worldwide survey on diabetes reveals that
among the entire diabetes cases more than 90% are account to type-II
[4]. e overall death rate in people with diabetes is about twice that of
people without diabetes [5].
e mechanism of alloxan action has been intensively studied,
predominantly in vitro, and is now characterized quite well. Using
isolated islets [6] and perfused rat pancreas [7] it was demonstrated
that alloxan evokes a sudden rise in insulin secretion in the presence
or absence of glucose. is phenomenon appeared just aſter alloxan
treatment and was not observed aſter repetitive exposure of islets to this
diabetogenic agent [6]. e sudden rise in blood insulin concentration
was also observed in vivo just aſter alloxan injection to rats [8]. Alloxan-
induced insulin release is, however, of short duration and is followed by
complete suppression of the islet response to glucose even when high
concentrations (16.6 mM) of this sugar were used [7]. e action of
alloxan in the pancreas is preceded by its rapid uptake by the β-cells
[9,10]. Rapid uptake by insulin-secreting cells has been proposed to be
one of the important features determining alloxan diabetogenicity.
e diabetogenic agent, alloxan, is a hydrophilic and chemically
unstable compound. e logarithm of the octanol/water partition
coefficient of alloxan was found to be -1.86; its half-life at pH 7.4
and 37°C in phosphate buffer was 1.5 min. e partition coefficients
and half-lives of the alloxan reduction products, alloxantin and
dialuric acid, were very similar to those of the parent compound;
N-methylalloxan and N,N’-dimethylalloxan were less hydrophilic but
more unstable. Alloxan and its N-methyl derivatives were reduced
by thiols and in the presence of glutathione and cysteine, rapid redox
cycling occurred, with formation of ‘active oxygen’ species [11]. On
the other hand, when a diabetogenic dose is used, the time of alloxan
decomposition is sufficient to allow it to reach the pancreas in amounts
that are deleterious [12].
Insulin therapy and oral hypoglycaemic agents offer effective
glycaemic control, but insulin therapy has shortcomings such as
ineffectiveness following oral administration, short shelf life, the need
for constant refrigeration, and fatal hypoglycaemia, in the event of excess
dosage [13]. As a result, there is a need to search for compounds with
effective antidiabetic activity when taken orally. e oral hypoglycemic
agents that are capable of reducing blood sugar level belong to two
chemical classes; sulfonylureas and biguanides [14].
However, the use of oral anti-diabetics is limited due to their adverse
side effects including hematological, cutaneous and gastrointestinal
*Corresponding author: : Bhaskar Sharma, Department of Biochemistry, Sam
Higginbottom Institute of Agriculture, Technology & Sciences , Allahabad- 211008,
U.P, India, Tel: +91-9696907157/+91- 8005319392; E-mail: : reserachpaper26@
gmail.com
Received August 17, 2013; Accepted October 19, 2013; Published October 24,
2013
Citation: Sharma B, Siddiqui S, Ram G, Chaudhary M, Sharma G (2013)
Hypoglycemic and Hepatoprotective Effects of Processed Aloe vera Gel in a
Mice Model of Alloxan Induced Diabetes Mellitus. J Diabetes Metab 4: 303.
doi:10.4172/2155-6156.1000303
Copyright: © 2013 Sharma B, et al. This is an open-access article distributed under
the terms of the Creative Commons Attribution License, which permits unrestricted
use, distribution, and reproduction in any medium, provided the original author and
source are credited.
Abstract
Diabetes mellitus is a heterogeneous disease characterized by altered cellular metabolism. So many traditional
herbs are being used by diabetic patients to control this disease. The aim of this study was to investigate the effects
of aqueous extract of aloe vera leaves on hypoglycemic activity and hepatoprotective effects after alloxan injection
in Swiss albino mice. In this study, aqueous leaf extract of aloe vera was carried out. Diabetes was induced in mice
by alloxan monohydrate at dose of 150 mg/kg body weight injected intraperitoneally. Also alloxanized induced mice
were administered with 300 and 500 mg/kg body weight orally daily of extract for a period of 21 days. At the end
of the administration period, the mice were anaesthesized and dissect for the collection of blood and liver tissues.
In diabetic mice, the SGOT, SGPT and bilirubin level as well as serum glucose levels were significantly increased
(p<0.05) in comparison with the control groups. Diabetic mice group treated by extract at the dose of 300 and 500
mg/kg body weight orally significantly (p<0.05) reduced and normalised these biochemical parameters compared
with alloxan induced diabetic group. Histopathological study also did show adverse alternation in the morphological
architecture of the liver tissue. The results suggested that aqueous extract of aloe vera leaves possesses protective
effect against alloxan induced diabetic mice.