FEMS Microbiology Letters 84 (1991) 15-22
© 1991 Federation of European Microbiological Societies 0378-1097/91/$03.50
Published by Elsevier
15
FEMSLE 04656
Growth, sporulation and enterotoxin production by Clostridium
perfringens Type A in the presence of human bile salts
Norma L. Heredia t,,, Ronald G. Labbe 3, Manuel A. Rodriguez
and Jose S. Garcia-Alvarado 2
1 Departamento de Microbiologia, Facultad de Medicina, and 2 Departamento de Microbiologia e lnmunologia, Facultad de Ciencias
Biologicas, Universidad Autonoma de Nuevo Leon, Monterrey, Mexico and ~ Food Science Department, University of Massachusetts,
Amherst MA 01003, U.S.A.
Received 5 August 1991
Accepted 6 August 1991
Key words: Clostridium perfringens; Bile salt; Enterotoxin; Sporulation
1. SUMMARY
The effect of human bile juice and bile salts
(sodium cholate, sodium taurocholate, sodium
glycochenodeoxycholate and sodium chenodeoxy-
cholate) on growth, sporulation and enterotoxin
production by enterotoxin-positive and entero-
toxin-negative strains of Clostridium perfringens
was determined. Each bile salt inhibited growth
to a different degree. A mixture of bile salts
completely inhibited the growth of enterotoxin-
positive strains of this organism. Human bile juice
completely inhibited growth of all the strains at a
dilution of 1 : 320. A distinct stimulatory effect of
the bile salts on sporulation was observed in the
Correspondence to: R.G. Labbe, Food Science Department,
University of Massachusetts, Amherst, MA 01003, U.S.A.
* Conacyt Fellow. Present address: Departamento de Micro-
biologia e Inmunologia Facultad de Ciencias Biologicas,
U.A.N.L. Apartado Postal 124-F, San Nicolas N.L. 66450,
Mexico.
case of C. perfringens strains NCTC 8239 and
NCTC 8679. The salts also increased enterotoxin
concentrations in the cell extracts of the entero-
toxin-positive strains tested. No effect on entero-
toxin production was detected when an entero-
toxin-negative strain was examined.
2. INTRODUCTION
Clostridium perfringens is one of the most im-
portant agents of human food-borne illness. This
illness is due to an enterotoxin produced by this
organism during sporulation in the small intes-
tine. Its symptoms, which include acute abdomi-
nal pain, diarrhea, and in some cases nausea and
vomiting, appear 6-8 h after the ingestion of food
contaminated with large numbers of vegetative
cells of C. perfringens. When the cells reach the
small intestine they sporulate and produce an
intracellular enterotoxin which is released into
the intestinal lumen where the spore is liberated
[1].
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