Arch Virol (t988) 99:9-19
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© by Springer-Verlag t 988
Acid-labile human interferon alpha production by peripheral blood
mononuclear cells stimulated by HIV-infected cells
M. R. Capobianchi, F. De Marco, P. Di Marco, and F. Dianzani
The Institute of Virology, University of Rome, Rome, Italy
Accepted December 18, 1987
Summary. We compared the properties of interferon (IFN) induced in peripheral
blood mononuclear cells (PBMC) by free infectious HIV to that induced by
HIV-infected cells fixed with glutaraldehyde. While the IFN induced by HIV
was a conventional IFN alpha, the IFN induced by HIV-infected cells, although
sharing with IFN alpha both antigenic properties and molecular weight, was
strongly inactivated by treatment at pH lower than 4. The ability to induce
acid-labile IFN alpha was exerted both by the chronically--infected cell line
H9/HIV and by normal PBMC infected in vitro with HIV, while infection of
inducers cells with viruses other than HIV made these cells capable of inducing
only acid-stable IFN alpha. The cell involved in the production of this type of
IFN seems to be B-lymphocyte.
Because the presence of acid-labile IFN alpha in the serum of AIDS patients
has been described, we suggest that this unusual IFN derives from interaction
between circulating B-lymphocytes and the HIV-infected cells.
Introduction
Acquired immunodeficiency syndrome (AIDS) is caused by infection with T-
cell tropic retroviruses known as HTLVIII-LAV, recently reclassified HIV [24].
These viruses are capable of selectively infecting CD4 + lymphocytes, but rep-
lication in other cell types has been reported, provided the target cells are
expressing CD4 antigen [10]. The disease is characterized by a profound defect
in cell-mediated immunity, as shown by decreased hypersensitivity skin reac-
tions, decreased response in vitro to antigenic and/or mitogenic stimulation,
decreased cytotoxic lymphocyte responses, polyclonal B-cell activation, de-
creased monocyte chemotaxis and a number of serologic abnormalities [15].
Among the serologic abnormalities, the presence of an unusual acid-labile in-
terferon (IFN) has been repeatedly found, and it is considered to be a marker
of infection with HIV evolving toward illness, as more than 80% of sera from
patients with full-blown AIDS or with AIDS-related complex (ARC) have been
found to display in their sera acid-labile IFN alpha [1, 6, 12, 19].