ELSEVIER
Nicotine-Haloperidol Interactions and
Cognitive Performance in Schizophrenics
Edward D. Levin, Ph.D., William Wilson, Ph.D., Jed E. Rose, Ph.D., and Joseph McEvoy, M.D.
Nearly 90% of schizophrenics smoke cigarettes,
considerably higher than the general population's rate of
25%. There is some indication that schizophrenics may
smoke as a form of self-medication. Nicotine has a variety of
pharmacologic effects that may both counteract some of the
cognitive deficits of schizophrenia and counteract some of
the adverse side effects of antipsychotic drugs. In the
current study, we assessed the interactions of haloperidol
and nicotine on cognitive performance of a group of
schizophrenics. These patients were in a double-blind study,
randomly assigning them to low, moderate, and high dose
levels of haloperidol. The subjects, all smokers, came to the
laboratory on four different mornings after overnight
deprivation from cigarettes. In a double-blind fashion, they
were administered placebo, low (7 mg/day), medium (14
mg/day), or high (21 mg/day) dose nicotine skin patches.
Three hours after administration of the skin patch, the
KEY WORDS: Nicotine; Haloperidol; Memory; Reaction
time; Schizophrenia
The very high rates of cigarette smoking among schizo-
phrenics, nearly 90%, has been noted in a variety of
studies (Hughes et al. 1986; O'Farrell et al. 1983). Over
the past 30 years, smoking in the general population in
the United States has declined to about 25% (Fiore
1992), but the smoking rates in certain groups including
From the Department of Psychiatry (EDL, WW, JER, JM) and
Department of Pharmacology (EDL), Duke University Medical Cen-
ter, Department of Research Service, VA Medical Center (JER),
Durham, North Carolina.
Address correspondence to: Dr. Edward D. Levin, Neurobehav-
ioral Research Laboratory, Department of Psychiatry, Box #3412,
Duke University Medical Center, Durham NC 27710.
Received August 21, 1995; revised September 28, 1995; accepted
January 5, 1996.
NEUROPSYCHOPHARMACOLOGY 1996-VOI.. 15, NO. 5
© 1996 American College of Neuropsychopharmacology
Published by Elsevier Science Inc.
655 Avenue of the Americas, New York, NY 10010
subjects were given a computerized cognitive test battery
including: simple reaction time, complex reaction time
(spatial rotation), delayed matching to sample, the
Sternberg memory test, and the Conners continuous
performance test (CPT). With the placebo nicotine patch,
there was a haloperidol dose-related impairment in delayed
matching to sample choice accuracy and an increase in
response time on the complex reaction time task. Nicotine
caused a dose-related reversal of the haloperidol-induced
impairments in memory performance and complex reaction
time. In the CPT, nicotine reduced the variability in
response that is associated with attentional deficit. These
results demonstrate the effects of nicotine in reversing some
of the adverse side effects of haloperidol and improving
cognitive performance in schizophrenia. © 1996
American College of Neuropsychopharmacology
[Neuropsychopharmacology 15:429-436, 1996]
schizophrenics remains high. Nicotine, the principal
psychoactive ingredient in tobacco, has a wide variety
of pharmacologic effects. Some of these effects such as
cognitive enhancement can be beneficial, despite the
other adverse health consequences of tobacco use. Some
smokers might use tobacco as a form of self-medication.
It seems most likely that groups who smoke most
heavily may be most likely to be engaging in self-medi-
cation. Several lines of evidence point to the likelihood
for nicotine self-medication in schizophrenics (Lohr and
Flynn 1992). The current study was conducted to deter-
mine the pharmacologic effects of nicotine on cognitive
function in schizophrenics, whether it might reverse
some of the cognitive deficits associated with schizo-
phrenia and as well as deficits that result from antipsy-
chotic medication.
Nicotine has been widely shown to be very effective
in stimulating dopamine (DA) release (Andersson et al.
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