Vol. 123, No. 3, 1984
September 28, 1984
BIOCHEMICAL AND BIOPHYSICALRESEARCH COMMUNICATIONS
Pages 1122-1129
T 3 STIMULATES THE SYNTHESIS OF A SPECIFIC mRNA IN PRIMARY HEPATOCYTE CULTURE.
Cary N. Marlash, Donald B. Jump and Jack H. Oppenheimer
Department of Medicine, Division of Endocrinology and Metabol Ism,
and Department of Genetics and Cell Biology,
University of Minnesota, Minneapolis, MN 55455
Received August 3, 1984
The mechanism of trIIodothyronlne (T 3) Induction of a thyroid hormone
respons!ve mRNA (mRNAs14) was studied In primary cultures of adult rat
hepatocytes. T3 Induced mRNAs14 in less than one hour after addltlon to the
cultures. After 24 hours exposure to T3, the level of mRNAs14 was 2.5 to 6
times above the untreated controls. Addition of Actinomycln-D to both Induced
and control cultures led to slmllar mRNAs14 disappearance curves, Implying
that T3 augments the synthesis of mRNAs14 rather than stablllzlng pre-formed
mature mRNA. Glucagon Inhibits the T 3 induction of mRNAs14. When added to
both induced and control cultures, glucagon leads to slmllar fractional decay
curves for mRNAs14, confirming the Actlnomycln-D studies. These findings
demonstrate T3 Induces mRNAs14 dlrectly In the hepatocyte by Increasing the
synthesis of the mature mRNA. © 1984Academic Press, Inc.
We have recently Identified a mRNA In rat Ilver which Is rapidly
responsive to the in vivo admlnlstratlon of T3, mRNAsI 4 (1-3). The mRNA codes
for a proteln of M r 17,500 and pl of 4.9 located in hepatic cytosol (3)°
Because Induction of thls mRNA occurs within 20 minutes following T3
admlnlstratlon, It may well represent a primary response to thyroid hormone.
However, additlonal data Indicate that factors other than thyroid hormone
regulate the level of mRNAs1 4. Thus, carbohydrate feeding also Induces a
response (4), an effect which appears to be as prompt as that noted with T 3
(unpublished observations). The level of mRNAs1 4 also undergoes a marked
clrcadlan varlatlon (3) due to as yet undefined factors. Moreover,
approximately one-third of the T 3 responsive hepatic mRNAts are not altered by
T3 directly, but rather, by pituitary growth hormone secretion stimulated by
T 3 (4). Therefore, It Is especial ly important to establ ish that T3 stimulates
the formation of mRNAs14 directly at the hepatocellular level.
An Isolated hepatocyte culture model Is Ideally suited to answer this
question, lye have previously used such a model to Investigate the synergism
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