Biomonitoring 2014; Volume 1: 25–38
Doi: 10.2478/bimo-2014-0003
Received April 3, 2014 accepted May 9, 2014
1 Introduction
Testicular cancer represents the most commonly diagnosed
cancer in men younger than 35. Its incidence in Europe is
on the rise, doubling every two decades [1]. The highest
rates of testicular cancer were noted for white Caucasian
populations in industrialised countries, mostly in western
and northern Europe and Australia/New Zealand [2].
Seminoma is the most common testicular tumour and is
believed to arise from the seminiferous epithelium of the
mature or maturing testis. Improvements in treatment
of testicular cancer have resulted in prolonged survival
and high cure rates. For patients with stage I seminoma
in particular, regular surveillance, radiotherapy, and/
or carboplatin-based chemotherapy are viable options
following an orchidectomy [3]. The prognosis for these
patients is good, irrespective of the choice of treatment
[4]. Radiotherapy is usually applied in patients with stage
I, as adjuvant treatment following a radical inguinal
orchidectomy, to minimize the potential relapse in the
retroperitoneum and/or ipsilateral pelvis. Furthermore, in
patients with stage IIA and IIB, where the regional lymph
nodal metastasis is smaller than 5 cm, radiotherapy is
given as a definitive therapeutic intervention. In stage
IIC, where the regional node metastasis exceeds 5 cm,
cisplatin-based combination chemotherapy is commonly
used as the primary form of treatment [5].
Here we report a case of a 34-year-old male patient
with a stage IB seminoma relapsed in the pelvis two
years after surgery and adjuvant radiotherapy. During
the second cycle of bleomycin-etoposide-cisplatin (BEP)
chemotherapy, he suffered a cerebral infarction. This
case is particularly interesting from the clinical point of
view because it describes a very rare cisplatin-related
Abstract: We report a case of a testicular seminoma patient
with relapse who was irradiated after acute cerebral
infarction induced by cisplatin-based chemotherapy.
Lymphocytic genome instability was studied using an
alkaline comet assay, analysis of structural chromosome
aberrations, and cytokinesis-block micronucleus assay
in blood samples collected before and after PET CT
scanning that preceded radiotherapy, as well as before the
administration of the first and after the administration of
the last fraction of 3D conformal radiation. A challenge-
test with hydrogen peroxide (H
2
O
2
) was performed on
isolated peripheral blood lymphocytes in order to establish
to what extent earlier therapies had modified the response
of the patient’s DNA to external stimuli with a genotoxic
chemical. Levels of primary DNA damage in lymphocytes
increased after diagnostic exposure, lowered prior to
administration of a conformal 3D radiotherapy, and were
the highest at the end of radiotherapy. Ex vivo exposure to
H
2
O
2
caused additional lymphocyte DNA damage, which
gradually increased 15 and 30 minutes after treatment.
Diagnostic and therapeutic exposure to radiation caused
measurable cytogenetic damage that was subjected
to extensive repair. All of the obtained results point to
increased genomic instability in the patient which should
be taken into account in his future medical surveillance.
Keywords: Cisplatin-based chemotherapy, chromosome
aberrations, comet assay, irradiation, lymphocyte,
micronucleus, testicular cancer
Case report Open Access
© 2014 Marija Gamulin et al., licensee De Gruyter Open. This work is licensed under the Creative Commons Attribution-NonCommercial-NoDerivs 3.0
License.
Marija Gamulin, Ana Pastuhović, Fedor Šantek, Mislav Grgić, Nevenka Kopjar*
Biomarkers used to assess radio- and
chemotherapy-induced lymphocyte genome
instability in a case of cerebral infarction during
relapse of a testicular seminoma
*Corresponding authors: Nevenka Kopjar: Institute for Medical Re-
search and Occupational Health, Mutagenesis Unit, Zagreb, Croatia,
Tel. +385 1 46 82 630; Fax +385 1 46 73 303, e-mail: nkopjar@imi.hr
Marija Gamulin, Fedor Šantek, Mislav Grgić: Department of
Oncology, Clinical Hospital Centre Zagreb, Zagreb, Croatia
Ana Pastuhović: School of Medicine, University of Zagreb, Zagreb,
Croatia
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