Prevention of Sudden Death in Hypertrophic
Cardiomyopathy
But Which Defibrillator for Which Patient?
Giuseppe Boriani, MD, PhD; Barry J. Maron, MD; Win-Kuang Shen, MD; Paolo Spirito, MD
C
ase presentation: A 14-year-
old girl died suddenly and un-
expectedly while exercising.
Clinical and echocardiographic family
screening identified hypertrophic car-
diomyopathy (HCM) in her only sib-
ling, an asymptomatic 17-year-old
boy. In her brother, echocardiography
showed extreme septal ventricular hy-
pertrophy, 35 mm in thickness; left
ventricular (LV) outflow obstruction
was absent. Left atrial dimension was
42 mm. Holter monitoring showed 2
runs of nonsustained ventricular
tachycardia (5 and 8 beats). Blood
pressure response during exercise was
normal. The patient was judged to be
at high risk for sudden death and a
candidate for an implantable
cardioverter-defibrillator (ICD). The
overall favorable clinical profile (ie, no
symptoms, preserved systolic function,
and low risk of developing atrial fibril-
lation [AF]) suggested that long-term
survival principally relied on effective
prevention of sudden death. Therefore,
ICD selection centered on the long-
term reliability of the ICD system,
particularly the leads. A single-
chamber ICD with a single-coil active-
fixation lead was implanted to facili-
tate extraction should lead removal be
required. Moreover, a high–shock out-
put ICD was used because of the mas-
sive LV hypertrophy and possible high
defibrillation threshold.
Background
During the past few years, interest has
increased in the use of the ICD in
genetic cardiac diseases associated
with sudden cardiac death, such as
HCM, long-QT syndrome, arrhythmo-
genic right ventricular cardiomyopa-
thy, and Brugada syndrome.
1,2
HCM is
by far the most common of these
cardiac conditions, with a prevalence
of about 1:500 in the general popula-
tion.
3,4
Because most HCM patients at
high risk for sudden death are young,
with no or only mild symptoms and
preserved systolic function, prevention
of sudden death by the ICD may pro-
long life substantially in this disease
and could offer a normal or near-
normal life expectancy to many pa-
tients.
1,4,5
Indeed, recent investigations
have shown that the ICD is effective in
preventing sudden death in HCM.
1,6
In
a multicenter study of high-risk HCM
patients, the device intervened appro-
priately and terminated ventricular
tachycardia or fibrillation at a rate of
5% per year for primary prevention
and 11% per year for secondary pre-
vention, over an average follow-up of
3 years.
1
The number of HCM patients
implanted with an ICD also has in-
creased substantially over the past few
years. Therefore, a large ICD cohort is
now emerging that differs from the
general ICD population because of
younger age, lower rate of life-
threatening events, and longer ex-
pected survival. Consequently, in such
patients, the selection of the most ap-
propriate ICD system with adequate
long-term reliability has become criti-
cally important, and the complexity of
such decisions is enhanced by the clin-
ical heterogeneity of HCM.
The 2002 American College of Car-
diology/American Heart Association/
North American Society of Pacing and
Electrophysiology consensus guide-
lines do not address the specific issue
of lead and device selection in HCM
and are largely based on trials per-
From the Istituto di Cardiologia, Università di Bologna, Azienda Ospedaliera S. Orsola-Malpighi, Bologna, Italy (G.B.); Hypertrophic Cardiomyopathy
Center, Minneapolis Heart Institute Foundation, Minneapolis, Minn (B.J.M.); Division of Cardiovascular Diseases and Internal Medicine, Mayo Clinic,
Rochester, Minn (W.-K.S.); and Divisione di Cardiologia, Ente Ospedaliero Ospedali Galliera, Genoa, Italy (P.S.).
Correspondence to Giuseppe Boriani, MD, PhD, Istituto di Cardiologia, Università di Bologna, Policlinico S. Orsola, Azienda Ospedaliera S.
Orsola-Malpighi, Via Massarenti 9, 40138, Bologna, Italy. E-mail cardio1@med.unibo.it
(Circulation. 2004;110:e438-e442.)
© 2004 American Heart Association, Inc.
Circulation is available at http://www.circulationaha.org DOI: 10.1161/01.CIR.0000144463.65977.C9
CLINICIAN UPDATE
e438
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