© Schattauer 2015 Thrombosis and Haemostasis 114.5/2015
876
Aspirin for primary prevention in diabetes mellitus: from the
calculation of cardiovascular risk and risk/benefit profile to
personalised treatment
Francesca Santilli
1
; Pasquale Pignatelli
2
; Francesco Violi
2
; Giovanni Davì
1
1
Department of Medicine and Aging, Center of Excellence on Aging, University of Chieti, Chieti, Italy;
2
I Clinica Medica, Sapienza University, Rome, Italy
Summary
Type 2 diabetes mellitus is characterised by persistent thromboxane
(TX)-dependent platelet activation, regardless of disease duration.
Low-dose aspirin, that induces a permanent inactivation of platelet
cyclooxygenase (COX)-1, thus inhibiting TXA
2
biosynthesis, should be
theoretically considered the drug of choice. The most up-to-date meta-
analysis of aspirin prophylaxis in this setting, which includes three
trials conducted in patients with diabetes and six other trials in which
such patients represent a subgroup within a broader population, re-
ported that aspirin is associated with a non-significant decrease in the
risk of vascular events, although the limited amount of available data
precludes a precise estimate of the effect size. An increasing body of
evidence supports the concept that less-than-expected response to as-
pirin may underlie mechanisms related to residual platelet hyper-reac-
tivity despite anti-platelet treatment, at least in a fraction of patients.
Among the proposed mechanisms, the variable turnover rate of the
drug target (platelet COX-1) appears to represent the most convincing
determinant of the inter-individual variability in aspirin response. This
review intends to develop the idea that the understanding of the de-
terminants of less-than-adequate response to aspirin in certain indi-
viduals, although not changing the paradigm of the indication to low-
dose aspirin prescription in primary prevention, may help identifying,
in terms of easily detectable clinical or biochemical characteristics, in-
dividuals who would attain inadequate protection from aspirin, and
for whom different strategies should be challenged.
Keywords
Antiplatelet agents, diabetes mellitus, aspirin response, prevention
Correspondence to:
Giovanni Davì
Center of Excellence on Aging
“G. D’Annunzio” University Foundation
Via Luigi Polacchi 11, 66013 Chieti, Italy
Tel: +39 0871 541312, Fax: +39 0871 541261
E-mail: gdavi@unich.it
Received: March 5, 2015
Accepted after major revision: May 29, 2015
Epub ahead of print: August 6, 2015
http://dx.doi.org/10.1160/TH15-03-0202
Thromb Haemost 2015; 114: 876–882
Viewpoint Article
Search strategy
This document is based on a comprehensive review of the avail-
able literature. PubMed database was searched through February
2015 for articles in English reporting aspirin use for primary pre-
vention in diabetes mellitus. The following search terms were
used: ‘aspirin’, ‘diabetes’, ‘primary prevention, ‘antiplatelet therapy’,
‘cardiovascular diseases’, ‘prophylaxis’, ‘cardiovascular risk’ and
‘guidelines’. Identified references were hand-searched to locate
other potentially useful references. Clinical randomised trials,
prospective cohort studies and retrospective analyses were in-
cluded, as well as meta-analyses; abstracts were excluded.
In the past two decades, preventive care for adults with diabetes de-
clined substantially the rates of diabetes-related complications (acute
myocardial infarction (MI), stroke, end-stage renal disease, lower-ex-
tremity amputation). However, a large burden of disease persists be-
cause of the continued increase in the prevalence of diabetes (1).
The latest American Diabetes Association (ADA) guidelines for
prevention and management of diabetes complications, suggest
considering aspirin therapy (75–162 mg/day) as a primary preven-
tion strategy in those with diabetes at increased cardiovascular risk
(10-year risk > 10 %). This includes most men aged > 50 years or
women aged > 60 years who have at least one additional major risk
factor (2).
Aspirin to prevent initial cardiovascular events
in diabetes mellitus: where do we stand?
In general populations without previous cardiovascular disease
(CVD), the absolute benefits of antiplatelet prophylaxis are of un-
certain value in primary prevention (3), because the balance be-
tween vascular events avoided and major bleeds caused by aspirin
is substantially uncertain. In a systematic review of six trials in-
cluding 95,000 individuals, a 12 % risk reduction (4) was found,
mainly due to a reduction in non-fatal MI, with a non statistically
significant net effect on stroke (a reduction in ischaemic stroke
and a slight increase in haemorrhagic stroke). Moreover, vascular
mortality was not changed by aspirin treatment. Major bleeding
(gastrointestinal and extracranial) increased by 0.03 % per year.
Two updated meta-analyses (5, 6), including three more recent
trials POPAPAD (7), JPAD (8), and AAA (9), demonstrated a
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