The Ayurvedic Perspective on the Etiopathogenesis of Neurodegeneration
Jibin Joy
*
Translational Scientific Research, Mundomthanath House, Poothrikka P.O, Puthencruz via Ernakulam, Kerala-682308, India
*
Corresponding author: Jibin Joy, Translational Scientific Research, Mundomthanath House, Poothrikka P.O, Puthencruz via Ernakulam, Kerala-682308, India, Tel:
+91-0484-2764941; E-mail: jibinjoyindia@gmail.com
Rec date: Sep 06, 2015; Acc date: Sep 26, 2015; Pub date: Sep 28, 2015
Copyright: © 2015 Jibin Joy. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use,
distribution, and reproduction in any medium, provided the original author and source are credited.
Abstract
The homeostatic signaling system in Homo sapiens is highly complex involving diverse physiological processes
which are what enables the biological regulation of life. Human body is a highly dynamic system, the normal
functioning of which is coordinated and maintained by a well-established mechanism of homeostatic regulation.
Disorder genesis and progression has an underlying disruption in homeostatic regulation, even though
pathophysiology and clinical presentations may vary. Progressive neurodegenerative conditions are an outcome of
disrupted homeostatic regulation, which triggers accumulation of dysfunctions and impairment of neuroplasticity.
Ayurveda considers neurodegenerative conditions to be brought by the imbalance of 'Vata', the energy that
moves through the brain and the nerves. The scholars behind classical ayurvedic literature considered neural
impulses to be a kind of air traveling through the body, controlling both voluntary and involuntary functions. Oxygen
is the real time mediator of homeostatic signaling system and defective oxygen transport; thereby impairment in
oxygenation is the prime underlying factor in neurodegenerative conditions. The broad spectrum therapeutic effects
demonstrated by Withania Somnifera or Ashwagandha are a result of its profound mechanism of enhancement of
oxygenation. Also the ayurvedic branch of medicine known as 'Rasa Shastra' uses 'Bhasma', traditionally prepared
Nano medicines, some of which shows influential enhancement in oxygenation.
Keywords: Neurodegeneration; Synaptogenesis; Alzheimer’s disease;
Homeostatic regulation; Parkinson's disease
Abbreviations
COX: Cytochrome C Oxidase; Oxphos-Oxidative phosphorylation;
NFT: Neuro fbrillary Tangles; AD: Alzheimers Disease; PD:
Parkinsons Disease; DLB: Dementia Lewy Body; CNS: Central
Nervous System; Gpx: Erythrocyte glutathione peroxidase; SOD:
Superoxide dismutase; CAT: Catalase; MB: Methylene Blue
Introduction
Te classical ayurvedic literatures on 'Vata Raktha Chikitsa' [1]
clearly suggests that complex disorders afecting nervous system
originates from an underlying imbalance of 'vata'. Furthermore,
Ayurveda considers human body as a vehicle for spiritual experience
which is suggestive of the fact that human consciousness is not locally
stored in nervous system. Tis co-relates with the studies on the
underlying mechanisms of cytoskeletal signaling [2]. It has been
favored by quantum theory perspectives, that fundamental
disturbances in consciousness arises from an underlying disturbance in
cytoskeletal system. Te micro tubule based concepts of consciousness
has been validated in several clinical research studies and implicate
aberrant micro tubules as a pathogenetic mechanism in disorders
afecting consciousness [3-5]. Te cytoskeletal system exhibits dynamic
behavior, and it does interacts with hundreds of associated proteins,
molecular motors, cross linkers. Also involved in many cell signalling
pathways and cytokinesis [6-8]. As described in classical ayurvedic
literature, when it comes to the fve elements of creation, air is
considered to be related to movement and electrical energy. And hence
it is suggestive of the fact that oxygen is the fundamental electron
transport mechanism in the universe. It’s the way through which the
universe is monitoring, recording, maintaining internal stability,
obtaining coordinated responses and is managing the ecology of life.
All processes of integration and coordination of function in the
ecology of life, whether mediated by nervous and hormonal systems
comes under homeostatic regulation. Te primary target and aim of
every disease and disorder in the ecology of life is to impair
oxygenation the outcome of which is the disruption of homeostatic
regulation. An illness or disease thrives only in an oxygen defcient
environment. Impairment in oxygenation implies disruption of
homeostatic regulation [9]. Furthermore therapeutic approaches used
in ayurvedic medicinal systems for longevity, vitality are suggestive of
the underlying mechanism of enhancement of oxygenation. Withania
Somnifera or 'Ashwagandha' is showing a similar therapeutic potential.
Also certain nanomedicines used in ayurvedic medicinal system
prepared in traditional manner which are referred to as 'Bhasma' are
demonstrating similar potential [10].
Since oxygen is the mediator of electron–transport mechanism, a
dysfunction in the electron–transport mechanism is suggestive of
disrupted homeostatic regulation. And a lot of experimental and
clinical studies favor a major disruption in homeostatic regulation in
neurodegenerative conditions. Respiratory chain defcits,
mitochondria dysfunction, exacerbated reactive oxygen species
production, low hemoglobin levels, defciency in the terminal complex
of the mitochondrial electron transport chain-cytochrome c oxidase
(COX), defects in oxidative phosphorylation (Oxphos) has been
observed in the pathophysiology of neurodegenerative conditions.
Tese observations suggests that impairment in oxygenation is at the
core of neurodegeneration. Its quite interesting that a rejected theory
on neurodegeneration, which had considered 'circle of willies-
atherosclerosis' as a contributing factor to Alzheimer's disease, neuritic
Joy J Health Edu Res Dev 2015, 3:3
DOI: 10.4172/2380-5439.1000137
Commentary Open Access
J Health Edu Res Dev
ISSN:2380-5439 JHERD, an open access journal
Volume 3 • Issue 3 • 1000137
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