Errors of Fact and Reasoning
in Consideration of
Shaken Baby Syndrome
To the Editor:
M
iller and Miller
1
reanalyze data from
others to suggest that male sex in-
creases the risk of subdural hemorrhage
after birth or head injury in infants and
children. As pediatric intensivists involved
daily in the care of severely head-injured
children, we are aware of fundamental
errors in the analysis and interpretation
that undermine this finding.
Their putative evidence is a recorded
excess of males with subdural bleeding
from a variety of causes. Any comment
about a male preponderance in subdural
bleeding has to consider a male prepon-
derance in the incidence of injury. Only
if subdural bleeding occurs in a higher
proportion of male children after severe
head injury (rather than simply numeri-
cally more children) can their hypothesis
be sustained. It is regrettable that despite
117 references, neither Miller and Miller
1
nor the reviewers thought it appropriate
to consider the unambiguous data that
there is a very significant male prepon-
derance in severe head injury at all ages.
The seminal article of Tilford et al,
2
ana-
lyzing 98,023 critically head-injured chil-
dren, describes 69,150 males (70.5%).
Similar proportions are evident in the
French data set (67% of 585 cases)
3
and
the UK Paediatric Intensive Care Audit
data (69% of 620 cases).
4
These data com-
bined give us 69,957/99,223 = 70.5% male
preponderance. This difference remains in
the youngest age groups.
4
Miller and Miller provide tables de-
scribing the absolute number of cases by
sex of subdural bleeding after accidental
(Table 2; 62.2%, males 163/262) and
inflicted trauma (Table 3; 62.6% males,
1007/1609). If we assume that their fig-
ures are correctly extracted from the lit-
erature then it is a simple matter of
estimation of the difference between pro-
portions in comparison to the data previ-
ously mentioned. We observe an absolute
risk reduction for subdural bleeding with
male sex of around 7% in both scenarios
(95% confidence interval, 2%Y13% for
accidental [P = 0.003] and 5%Y9% for
inflicted injury [P G 0.0002]; significance
of difference between 2 independent pro-
portions). Put simply, there is no evidence
that a higher proportion of male children
develop subdural hemorrhages than would
be expected from the baseline trauma epi-
demiology. Miller and Miller’s hypothesis
therefore can be refuted.
The situation should not be confused
by considering birth-related subdural bleed-
ing where head size may indeed contribute
to a differential level of trauma during
childbirth (including subdural bleeding) in
male neonates.
Unfortunately, the article is further
weakened by misquotations of the litera-
ture in describing subdural hemorrhage
frequency. For example, of the 4 largest
series quoted in Table 3, figures extracted
from 3 of these series are incorrect. The
figures presented from Wells et al
5
are
those for all types of intracranial hemor-
rhage (including 80 cases with epidural
hemorrhage, 36 with subarachnoid, and
13 with intraventricular bleeding) rather
than subdural bleeding as described. The
Canadian data are similarly misquoted in
that 364 cases are presented by Miller
and Miller, whereas the number with
subdural bleeding is presented as 313 in
the Canadian article.
6
The article of
Jenny et al
7
suffers the same fate as that of
Miller and Miller, incorrectly analyzing
173 cases (the number of head injuries)
not of cases with subdural hematomas
(n = 150).
Perhaps the most extraordinary ele-
ment of this article is the interpretation
applied to these flawed data. The authors
suggest (Fig. 1) that slowly enlarging sub-
dural hemorrhages in at-risk cases under-
lie what is misdiagnosed as shaken baby
syndrome. This is inconsistent with the
typical history of acute collapse, the im-
aging of thin-film subdural hemorrhages
(not space-occupying hematoma),
8
and
the pathology of the triad of predomi-
nantly diffuse hypoxic-ischemic encepha-
lopathy.
9
The authors themselves restate
these features.
8,9
Articles such as this that lack preci-
sion in data collection and analysis and
then build dramatic interpretations on these
flawed analyses do not advance knowledge.
Worse, they may contribute to misdiagnosis
of inflicted head injury, thereby exposing
vulnerable children to risk.
AUTHOR DISCLOSURE
INFORMATION
The authors declare no conflicts of
interest.
Joe Brierley, MBChB, MRCP
FRCPCH, MA
Mark J. Peters, MBChB, MRCP
FRCPCH, PhD
Paediatric Intensive Care Unit
Great Ormond St Hospital
London, UK
brierj@gosh.nhs.uk
REFERENCES
1. Miller R, Miller M. Overrepresentation of
males in traumatic brain injury of infancy
and in infants with macrocephaly: further
evidence that questions the existence of
shaken baby syndrome. Am J Forensic
Med Pathol. 2010;31(2):165Y173.
2. Tilford JM, Aitken ME, Anand KJ, et al.
Hospitalizations for critically ill children
with traumatic brain injuries: a longitudinal
analysis. Crit Care Med. 2005;33(9):2074Y2081.
3. Ducrocq SC, Meyer PG, Orliaguet GA, et al.
Epidemiology and early predictive factors
of mortality and outcome in children with
traumatic severe brain injury: experience of a
French pediatric trauma center. Pediatr Crit
Care Med. 2006;7(5):461Y467.
4. Parslow RC, Morris KP, Tasker RC, et al.
Epidemiology of traumatic brain injury in
children receiving intensive care in the UK.
Arch Dis Child. 2005;90(11):1182Y1187.
5. Wells RG, Vetter C, Laud P.Intracranial
hemorrhage in children younger than
3 years: prediction of intent. Arch Pediatr
Adolesc Med. 2002;156(3):252Y257.
6. King WJ, MacKay M, Sirnick A. Shaken
baby syndrome in Canada: clinical
characteristics and outcomes of hospital
cases. CMAJ. 2003;168(2):155Y159.
7. Jenny C, Hymel KP, Ritzen A, et al.
Analysis of missed cases of abusive head
trauma. JAMA. 1999;281(7):621Y626.
8. Geddes JF, Vowles GH, Hackshaw AK,et al.
Neuropathology of inflicted head injury in
children. II. Microscopic brain injury in
infants. Brain. 2001;124(Pt 7):1299Y1306.
9. Geddes JF, Hackshaw AK, Vowles GH,et al.
Neuropathology of inflicted head injury in
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RESPONSE
Reply:
I
n response to the letter by Peters and
Brierley regarding our study, we offer the
following comments
1
: Their main criti-
cism is the control group that we used.
We compared our results of male/female
LETTER TO THE EDITOR
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