genes
G C A T
T A C G
G C A T
Review
Gene Amplification and the Extrachromosomal Circular DNA
Noriaki Shimizu
Citation: Shimizu, N. Gene
Amplification and the
Extrachromosomal Circular DNA.
Genes 2021, 12, 1533. https://doi.org/
10.3390/genes12101533
Academic Editors: Eishi Noguchi and
Maciej Wnuk
Received: 7 August 2021
Accepted: 23 September 2021
Published: 28 September 2021
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Graduate School of Integrated Sciences for Life, Hiroshima University, 1-7-1 Kagamiyama,
Higashi-Hiroshima 739-8521, Hiroshima, Japan; shimizu@hiroshima-u.ac.jp
Abstract: Oncogene amplification is closely linked to the pathogenesis of a broad spectrum of human
malignant tumors. The amplified genes localize either to the extrachromosomal circular DNA, which
has been referred to as cytogenetically visible double minutes (DMs), or submicroscopic episome,
or to the chromosomal homogeneously staining region (HSR). The extrachromosomal circle from
a chromosome arm can initiate gene amplification, resulting in the formation of DMs or HSR, if it
had a sequence element required for replication initiation (the replication initiation region/matrix
attachment region; the IR/MAR), under a genetic background that permits gene amplification. In this
article, the nature, intracellular behavior, generation, and contribution to cancer genome plasticity of
such extrachromosomal circles are summarized and discussed by reviewing recent articles on these
topics. Such studies are critical in the understanding and treating human cancer, and also for the
production of recombinant proteins such as biopharmaceuticals by increasing the recombinant genes
in the cells.
Keywords: gene amplification; extrachromosomal DNA; double minutes; micronucleus; cancer;
genome plasticity; chromothripsis; gene expression; repeat-induced gene silencing
1. Gene Amplification and the Extrachromosomal Circles in Human Cancer
The amplification of oncogenes or drug-resistant genes plays a pivotal role in human
cell malignant transformation by conferring growth advantage to the cells through the
overproduction of the amplified gene product. A classical cytogenetic study located the
amplified genes at the extrachromosomal double minutes (DMs) or the chromosomal
homogeneously staining region (HSR) [1]. DMs and HSR mutually interconvert [2,3], and
share the same sequence [4]. DMs are stable extrachromosomal elements that contain
circular DNA. Circularity has been suggested based on electron microscopy [5], sensitivity
to radiation-mediated breakage [6], and the absence of telomeric structures [7]; this was
recently re-enforced by integrating ultrastructural imaging, long-range optical mapping,
and computational analysis of whole-genome sequencing [8]. In contrast, cytogenetically
undetectable circular DNA has been identified in many normal and cancer cell lines and
normal tissues more than three decades ago [9]. Recently, many reports have described
circular extrachromosomal DNA in normal or cancer cells [10]. In general, the circles
in normal cells [11,12] were smaller in size (less than 1 kbp) than those in cancer cells
(1–2 Mbp) [13]. The former is referred to as extrachromosomal closed circular DNA
(eccDNA), and the latter are referred to as extrachromosomal DNA (ecDNA). EcDNAs
are equivalent to conventional DMs; however, the term ecDNA was recently used instead
of DMs because it does not always appear as a doublet among the chromosome spread
specimens. Several extensive studies that used a large number of clinical samples together
with the most advanced techniques, unambiguously, reinforced the tight relationship
between malignancy and the appearance of ecDNA/DMs [13,14].
It is important to note that gene expression from the same amplicon sequence is
higher in the extrachromosomal context than in the chromosomal context [15] because
the chromatin of extrachromosomal DNA is more favorable for gene expression [8,16].
Consistently, DMs were replicated early in the S phase, while the HSRs of the same
Genes 2021, 12, 1533. https://doi.org/10.3390/genes12101533 https://www.mdpi.com/journal/genes