Téllez‑Navarrete et al. Mol Med (2021) 27:76
https://doi.org/10.1186/s10020‑021‑00320‑4
RESEARCH ARTICLE
Anti‑tuberculosis chemotherapy alters
TNFR2 expression on CD4+ lymphocytes
in both drug‑sensitive and ‑resistant
tuberculosis: however, only drug‑resistant
tuberculosis maintains a pro‑infammatory
profle after a long time
Norma A. Téllez-Navarrete
1
, Lucero A. Ramon-Luing
1
, Marcela Muñoz-Torrico
2
, Mario Preciado-García
1
,
Karen Medina-Quero
3
, Rogelio Hernandez-Pando
4
and Leslie Chavez-Galan
1*
Abstract
Background: Tuberculosis (TB) is an infectious disease. During TB, regulatory T cells (Treg) are related to poor prog-
nosis. However, information about conventional and unconventional Treg (cTreg and uTreg, respectively) is limited.
The tumour necrosis factor (TNF) and its receptors (TNFR1 and TNFR2) are necessary for mycobacterial infection, and
TNFR2 signalling is required to maintain Treg.
Methods: A blood sample of drug-susceptible (DS-TB) and drug-resistant tuberculosis (DR-TB) patients was obtained
before (basal) and after 2 and 6 months of anti-TB therapy. Expression of TNF, TNFR1, and TNFR2 (transmembrane form,
tm) on cTreg, uTreg, activated CD4+ (actCD4+), and CD4+ CD25- (CD4+) T cell subpopulations were evaluated. The
main objective was to identify immunological changes associated with sensitive/resistant Mtb strains and with the
use of anti-TB therapy.
Results: We found that after 6 months of anti-TB therapy, both DS- and DR-TB patients have decreased the frequency
of cTreg tmTNF+, CD4+ tmTNFR1+ and CD4+ tmTNFR2+. Nevertheless, after 6 months of therapy, only DR-TB
patients decreased the frequency of actCD4+ tmTNF+ and actCD4+ tmTNFR2+, exhibited a systemic inflammatory
status (high levels of TNF, IFN-γ and IL-12), and their purified CD4+ T cells showed that TNF and TNFR2 are up-regu-
lated at the transcriptional level. Moreover, DS- and DR-TB down-regulated TNFR1 and other proteins associated with
Treg (FOXP3 and TGFβ1) in response to the anti-TB therapy.
Conclusion: These results partially explain the differences in the immune response of DS-TB vs DR-TB. The frequency
of actCD4+ tmTNFR2+ cells and inflammatory status should be considered in the follow-up of therapy in DR-TB
patients.
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Open Access
Molecular Medicine
*Correspondence: lchavez_galan@iner.gob.mx
1
Laboratory of Integrative Immunology, Instituto Nacional de
Enfermedades Respiratorias “Ismael Cosío Villegas”, Calzada de Tlalpan No.
4510, CP. 14080 Mexico City, Mexico
Full list of author information is available at the end of the article