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Risk factors for treatment-limiting toxicities in
patients starting nevirapine-containing
antiretroviral therapy
Anouk M. Kesselring
a
, Ferdinand W. Wit
b
, Caroline A. Sabin
c
,
Jens D. Lundgren
d
, M. John Gill
e
, Jose M. Gatell
f
, Andri Rauch
g
,
Julio S. Montaner
h
, Frank de Wolf
a
, Peter Reiss
b
, Amanda Mocroft
c
,
on behalf of the Nevirapine Toxicity Multicohort Collaboration
Background: This collaboration of seven observational clinical cohorts investigated
risk factors for treatment-limiting toxicities in both antiretroviral-naive and experienced
patients starting nevirapine-based combination antiretroviral therapy (NVPc).
Methods: Patients starting NVPc after 1 January 1998 were included. CD4 cell count at
starting NVPc was classified as high (>400/ml/>250/ml for men/women, respectively) or
low. Cox models were used to investigate risk factors for discontinuations due to
hypersensitivity reactions (HSR, n ¼ 6547) and discontinuation of NVPc due to treat-
ment-limiting toxicities and/or patient/physician choice (TOXPC, n ¼ 10 186). Patients
were classified according to prior antiretroviral treatment experience and CD4 cell
count/viral load at start NVPc. Models were stratified by cohort and adjusted for age,
sex, nadir CD4 cell count, calendar year of starting NVPc and mode of transmission.
Results: Median time from starting NVPc to TOXPC and HSR were 162 days [inter-
quartile range (IQR) 31–737] and 30 days (IQR 17–60), respectively. In adjusted Cox
analyses, compared to naive patients with a low CD4 cell count, treatment-experienced
patients with high CD4 cell count and viral load more than 400 had a significantly
increased risk for HSR [hazard ratio 1.45, confidence interval (CI) 1.03–2.03] and
TOXPC within 18 weeks (hazard ratio 1.34, CI 1.08–1.67). In contrast, treatment-
experienced patients with high CD4 cell count and viral load less than 400 had no
increased risk for HSR 1.10 (0.82 – 1.46) or TOXPC within 18 weeks (hazard ratio 0.94,
CI 0.78–1.13).
Conclusion: Our results suggest it may be relatively well tolerated to initiate NVPc in
antiretroviral-experienced patients with high CD4 cell counts provided there is no
detectable viremia. ß 2009 Wolters Kluwer Health | Lippincott Williams & Wilkins
AIDS 2009, 23:1689–1699
Keywords: CD4 cell count, HIV, nevirapine, toxicity, viral load
Introduction
Nevirapine is frequently used as part of combination
antiretroviral therapy (cART) regimens, and is currently
listed as one of the alternative options for cART in
treatment-naive patients [1]. Nevirapine may also be used
in patients with prior cART experience, for example, to
minimize diarrhea or to reduce cardiovascular risks
a
HIV Monitoring Foundation,
b
Center for Poverty-Related Communicable Diseases, and Department of Infectious Diseases,
Tropical Medicine and AIDS, Academic Medical Center, University of Amsterdam, Amsterdam, the Netherlands,
c
University
College London Medical School, Royal Free Campus, London, UK,
d
Rigshospitalet and University of Copenhagen, Copenhagen
HIV Programme, Copenhagen, Denmark,
e
University of Calgary, Calgary, Canada,
f
Infectious Diseases & AIDS Units, Hospital
Clinic, University of Barcelona, Spain,
g
Division of Infectious Diseases, University Hospital Bern and University of Bern, Bern,
Switzerland, and
h
British Columbia Centre for Excellence in HIV/AIDS, St. Paul’s Hospital, Vancouver, BC, Canada.
Correspondence to Anouk M. Kesselring, MD, HIV Monitoring Foundation, Meibergdreef 9, 1105 AZ, Amsterdam, the
Netherlands.
Received: 22 January 2009; revised: 20 April 2009; accepted: 21 April 2009.
DOI:10.1097/QAD.0b013e32832d3b54
ISSN 0269-9370 Q 2009 Wolters Kluwer Health | Lippincott Williams & Wilkins
1689