Anti-vascular Endothelial Growth Factor for Choroidal Neovascularization
Associated with Toxoplasmosis: A Case Series
Farzan Kianersi
1
, Zahra Naderi Beni
2
, Afsaneh Naderi Beni
2*
, Heshmatollah Ghanbari
1
and Mostafa Ahmadi
2
1
Department of Ophthalmology, Isfahan University of Medical Science, Isfahan, Iran
2
Shahrekord University of Medical Sciences, Shahrekord, Iran
*
Corresponding author: Afsaneh Naderi Beni, Shahrekord University of Medical Sciences, Shahrekord, Iran, Tel: +98-381-2224400; E-mail: a_naderibeni@yahoo.com
Received date: June 17, 2015, Accepted date: August 21, 2015, Published date: August 31, 2015
Copyright: © 2015 Kianersi F, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted
use, distribution, and reproduction in any medium, provided the original author and source are credited.
Abstract
The purpose of the study was to evaluate the efficacy and safety of intravitreal anti-vascular endothelial growth
factor (anti-VEGF) in treatment of choroidal neovascularization (CNV) secondary to toxoplasmic retinochoroiditis in
five patients. After six months the CNV resolved as confirmed by fluorescein angiography and optical coherence
tomography (OCT) in all patients. The visual acuity improved from mean 20/400 to 20/80 and Central Macular
Thickness (CMT) decreased from 390 μm to 253 μm, which was maintained till the last follow-up visit.
Keywords: Anti-vascular endothelial growth factor; Choroidal
neovascularization; Toxoplasmosis
Introduction
Treatment modalities for choroidal neovascularization (CNV) due
to ocular toxoplasmosis include laser photocoagulation, surgery,
corticosteroids, and verteporfin therapy. Intravitreal injection of Anti-
vascular endothelial growth factor (anti-VEGF ) in the treatment of
CNV due to other conditions appears to be an effective and safe
therapeutic option. We report 5 cases of successful treatment with
anti-VEGF for symptomatic CNV due ocular toxoplasmosis.
Methods
The Isfahan University Medical Center approved this study. Our
study population consisted of 5 patients with CNV due to ocular
toxoplasmosis (3 women, 2 men) with a mean (SD) age of 27.2 (7.1)
years, all with documented notes suggested of clinical and serological
diagnosis of toxoplasmosis retinichoriditis and had been treated with
oral sulfadiazine and pyrimethamine, along with systemic steroids at
least 6 months before referral to our clinic.
All patients received a complete ophthalmologic evaluation. The
anterior chamber and vitreous cavity were quiet in all patients. No
signs suggestive of age-related macular degeneration were found.
Fluorescein (FA) criteria included evidence of leakage caused by CNV
(100% classic without an occult component) secondary to ocular
toxoplasmosis and presence of intraretinal or subretinal fluid
documented by optical coherence tomography (OCT). Intravitreal
anti-VEGF treatment was recommended for all patients (bevacizumab,
1.25 mg for 4 eyes and ranibizumab 0.5 mg for one eye).
Results
CNV was inactive in all eyes after Intravitreal anti-VEGF
treatments with an absence of leakage demonstrated by fluorescein
angiography, complete disappearance of exudative signs and reduction
of retinal thickness shown by OCT (Figure 1).
Figure 1: Fluorescein angiography (FA) and OCT before and after
treatment in 5 cases with choroidal neovascularization associated
with toxoplasmosis.
The mean follow-up was 14.4 months (range 6–24 months). Pre-
injection visual acuity measured 20/250 or worse in all eyes. According
to the last follow-up examination, visual acuity measured 20/30,
20/200, 20/200, 20/100 and 20/100 in eyes (Table 1). Mean Central
macular thickness (CMT) at baseline was 399 μm (380-410 μm). At the
last check, mean CMT reduction was 149 μm (65-175 μm), with a
mean thickness of 253 μm (range, 215-300 μm) (p=0.0002).
Kianersi et al., J Clin Exp Ophthalmol 2015, 6:4
DOI: 10.4172/2155-9570.1000463
Short Communication Open Access
J Clin Exp Ophthalmol
ISSN:2155-9570 JCEO, an open access journal
Volume 6 • Issue 4 • 1000463
Journal of Clinical & Experimental
Ophthalmology
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ISSN: 2155-9570