Copyright © 2018, the American Society of Anesthesiologists, Inc. Wolters Kluwer Health, Inc. Unauthorized reproduction of this article is prohibited.
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Anesthesiology, V 128 • No 4 710 April 2018
A
CUTE kidney injury (AKI) after coronary artery
bypass grafting (CABG) with extracorporeal circula-
tion (ECC) occurs in approximately one third of patients in
most institutions, including our own,
1
and leads to increased
long- and short-term morbidity and mortality.
1
Te source
for AKI in CABG is multifactorial, but renal ischemia–
reperfusion injury induced by the use of ECC is at least
part of the cause,
2,3
especially in the poorly oxygenated and
metabolic active outer medulla. A suggested mechanism is
induced mitochondrial damage through the opening of the
mitochondrial permeability transition pore (mPTP) dur-
ing reperfusion, leading to cell injury or death.
4,5
Animal
What We Already Know about This Topic
• Acute kidney injury is common after cardiac surgery with
cardiopulmonary bypass
• Animal studies suggest that cyclosporine may be protective
What This Article Tells Us That Is New
• In a double-blind trial, 154 cardiac surgical patients were
randomly assigned to 2.5 mg/kg cyclosporine or placebo
• Plasma cystatin C, a marker of renal injury, increased more in
patients given cyclosporine
• Cyclosporine does not reduce the risk of acute renal injury
after cardiac surgery
Copyright © 2018, the American Society of Anesthesiologists, Inc. Wolters Kluwer Health, Inc. All Rights Reserved. Anesthesiology 2018; 128:710-7
ABSTRACT
Background: Acute kidney injury is a common complication after cardiac surgery, leading to increased morbidity and mortal-
ity. One suggested cause for acute kidney injury is extracorporeal circulation–induced ischemia–reperfusion injury. In animal
studies, cyclosporine has been shown to reduce ischemia–reperfusion injury in the kidneys. We hypothesized that administer-
ing cyclosporine before extracorporeal circulation could protect the kidneys in patients undergoing cardiac surgery.
Methods: Te Cyclosporine to Protect Renal Function in Cardiac Surgery (CiPRICS) study was an investigator-initiated, double-
blind, randomized, placebo-controlled, single-center study. Te primary objective was to assess if cyclosporine could reduce acute
kidney injury in patients undergoing coronary artery bypass grafting surgery with extracorporeal circulation. In the study, 154 patients
with an estimated glomerular fltration rate of 15 to 90 ml · min
–1
· 1.73 m
–2
were enrolled. Study patients were randomized to receive
2.5 mg/kg cyclosporine or placebo intravenously before surgery. Te primary endpoint was relative plasma cystatin C changes from the
preoperative day to postoperative day 3. Secondary endpoints included biomarkers of kidney, heart, and brain injury.
Results: All enrolled patients were analyzed. Te cyclosporine group (136.4 ± 35.6%) showed a more pronounced increase from baseline
plasma cystatin C to day 3 compared to placebo (115.9 ± 30.8%), diference, 20.6% (95% CI, 10.2 to 31.2%, P < 0.001). Te same pat-
tern was observed for the other renal markers. Te cyclosporine group had more patients in Risk Injury Failure Loss End-stage (RIFLE)
groups R (risk), I (injury), or F (failure; 31% vs. 8%, P < 0.001). Tere were no diferences in safety parameter distribution between groups.
Conclusions: Administration of cyclosporine did not protect coronary artery bypass grafting patients from acute kidney
injury. Instead, cyclosporine caused a decrease in renal function compared to placebo that resolved after 1 month. (ANESTHE-
SIOLOGY 2018; 128:710-7)
This article is featured in “This Month in Anesthesiology,” page 1A. Supplemental Digital Content is available for this article. Direct URL
citations appear in the printed text and are available in both the HTML and PDF versions of this article. Links to the digital files are provided
in the HTML text of this article on the Journal’s Web site (www.anesthesiology.org).
Submitted for publication July 26, 2017. Accepted for publication December 27, 2017. From the Departments of Anesthesiology and
Intensive Care (P.E., A.D., E.G., B.B., C.M., A.E., L.A.) and Cardiothoracic Surgery (S.N., A.M., H.B.), Clinical Sciences, Lund University, Skåne
University Hospital, Lund, Sweden; the Department of Mitochondrial Medicine, Clinical Sciences, Lund University, Lund, Sweden (M.J.H.,
E.E.); the Frederik Meijer Heart and Vascular Institute, Spectrum Health, Grand Rapids, Michigan (S.J.); the Van Andel Institute, Grand Rapids,
Michigan (S.J.); and the Cardiovascular Institute, Stanford University, Stanford, California (S.J.).
Cyclosporine before Coronary Artery Bypass
Grafting Does Not Prevent Postoperative Decreases
in Renal Function
A Randomized Clinical Trial
Per Ederoth, M.D., Ph.D., Alain Dardashti, M.D., Ph.D., Edgars Grins, M.D., Björn Brondén, M.D., Ph.D.,
Carsten Metzsch, M.D., Ph.D., André Erdling, M.D., Shahab Nozohoor, M.D., Ph.D.,
Arash Mokhtari, M.D., Ph.D., Magnus J. Hansson, M.D., Ph.D., Eskil Elmér, M.D., Ph.D.,
Lars Algotsson, M.D., Ph.D., Stefan Jovinge, M.D., Ph.D., Henrik Bjursten, M.D., Ph.D.
PERIOPERATIVE MEDICINE
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