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S. Emonet
a
S. Hogendijk
a
J. Voegeli
b
P.A. Eigenmann
a, c
N. Roux
a
C. Hauser
a, d
a
Allergy Unit, Division of Immunology and
Allergy,
b
Department of Community Medicine and
c
Department of Pediatrics, University Hospital,
Geneva, and
d
Department of Dermatology and
Venereology of Western Switzerland,
Geneva/Lausanne, Switzerland
Case Report
Dermatology 1998;197:181–182
Ethanol-Induced Urticaria:
Elevated Tryptase Levels after
Double-Blind, Placebo-Controlled Challenge
Introduction
Ethanol may induce various kinds of
adverse skin reactions such as flushing and,
rarely, immediate-type hypersensitivity-like
reactions [1, 2]. Anaphylaxis related to eth-
anol intake appears to be exceptionally rare
[3, 4].
We report the case of a patient who pre-
sented urticaria after a double-blind, placebo-
controlled challenge test with ethanol and an
increased serum tryptase level.
Case Report
For 1 year, a 48-year-old man complained
about burning sensations in the hands and
feet a few minutes after ingestion of various
kinds of alcoholic but not following nonalco-
holic beverages. He reported that he had
recently started to experience itching and to
develop a rash on his face after every con-
sumption of even small amounts of alcohol.
He denied any other systemic symptom and
indicated to tolerate the ingestion of vinegar.
He had no personal history of atopy.
Skin prick tests to frequent aeroallergens
were negative. Darier’s sign was negative.
Skin prick tests with ethanol (9.6 and 96%)
and acetic acid (0.5 and 10% vol/vol) were
performed as described [4]. All tests were
negative in 3 healthy controls. The patient
tested negative except for a +++ reaction to
10% acetic acid. A double-blind, placebo-
controlled challenge with 20 ml ethanol 96%
(vol/vol) diluted in 300 ml of sparkling grape
juice with sugar as challenge vehicle and ve-
hicle alone was performed. It had previously
been confirmed that the ethanol-containing
juice could not be distinguished from the
juice without ethanol. Five minutes after in-
gestion of ethanol the patient developed a
burning sensation, facial erythema and, a few
minutes later, urticarial lesions on his chest
and upper arms. No dyspnea or hypotension
was noted. Serum tryptase determined by the
FEIA kit (Pharmacia, Uppsala, Sweden) be-
fore challenge was nondetectable, 3.8 U/ml
10 min after the challenge when urticarial
lesions appeared and again nondetectable
3 and 5 h after the challenge (detection limit
0.1 U/ml). The challenge with juice contain-
ing no ethanol was negative and not followed
by tryptase determination. The patient re-
fused further investigations and treatment.
Discussion
Adverse reactions to alcoholic beverages
are well known. Reactions may occur either
to constituents other than ethanol and to
ethanol itself. Besides drunkenness, the most
frequent reaction to ethanol is flushing.
Ethanol can accentuate or trigger flushing of
various etiology such as physiologic flushing
(erythema pudicum), postmenopausal flush-
ing, flushing due to rosacea and flushing due
to systemic disorders such as mastocytosis
or carcinoid syndrome. Ethanol has been
reported to trigger flushing in patients taking
a variety of drugs such as metronidazole,
griseofulvin, ketoconazole, chloramphenicol,
quinacrine, cefamandole, cefoperazone, mox-
alactam or calcium carbamide. Patients on
disulfiram medication are particularly prone
to flushing and other general symptoms, even
with very small amounts of ethanol. Ethanol
triggered flushing in patients taking chlor-
propamide appears to be dominantly inher-
ited [5] and can be prevented by acetyl sali-
cylic acid [6]. Flushing due to genetically
decreased activity of aldehyde dehydro-
genase isoforms is well known in Asians.
Besides flushing, ethanol intake can trigger
asthma attacks, as frequently observed in
Conrad Hauser
Allergy Unit, Hôpital Cantonal Universitaire
CH–1211 Geneva 14 (Switzerland)
Tel. +41 22 372 9381, Fax +41 22 372 9416
E-Mail hauser@cmu.unige.ch
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Key Words
Alcohol
Anaphylaxis
Tryptase
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Abstract
We present a 48-year-old patient who complained for 1 year about urticarial reactions which
appeared always when he ingested alcoholic beverages. Skin prick tests with ethanol were neg-
ative but positive with 10% acetic acid in the patient. Normal controls tested negative with
acetic acid. Skin prick tests to common immediate-type allergens were negative. The patient
underwent a double-blind, placebo-controlled challenge test. A few minutes after challenge
with ethanol but not with placebo, the patient developed erythema and wheals on the chest and
the upper arms. The tryptase serum level rose from undetectable (<0.1 U/ml) before challenge
to 3.8 U/ml after skin lesions had appeared. This case demonstrates that increased tryptase
serum levels can help in the diagnosis of ethanol-induced urticaria.
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Received: December 16, 1997
Accepted: March 16, 1998
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