acute intervention, 14-30 days subacute, and >30 days chronic. Complicated dissection was defined as rapidly expanding dissection, aortic rupture, end-organ malperfu- sion, or uncontrollable hypertension or pain. Statistical analysis was performed using SPSS with anal- ysis of long term survival by Kaplan-Meir. Results: 127 patients (74% men) underwent endovascular repair for type B aortic dissection between January 2006 and December 2017. Mean age of patients was 65.4 (SD 11.4) years. 53 repairs were carried out in the acute setting, 20 in subacute, and 54 in chronic. 41 (32.3%) were for uncompli- cated dissection. The proportion of repairs performed for un- complicated dissection decreased during the study period; 26/ 57 in 2006-2011 compared with 15/70 in 2012-2017. In-hos- pital mortality was 9.4% (11.3% for acute, 10.0% for subacute, and 7.4% for chronic). In-hospital mortality for uncomplicated dissection was 4.9% compared with 11.6% for complicated. None of the differences in mortality reached statistical signif- icance. After a median follow-up of 2.3 years (IQR 0.7-5.1), one year survival was 83.8% and 3-year survival 70.1%. Conclusion: The present series demonstrates comparable in hospital mortality rates in patients treated for type B aortic dissection, regardless of the timing of the intervention, and good longer term survival after the index aortic interven- tion. This highlights the importance of planning an index repair strategy that “future proofs” the aorta, facilitating subsequent interventions that are likely to be required over the patient ’ s lifetime. P-183 Type 2 Diabetes and Abdominal Aortic Aneurysm: Association Between Insulin Resistance Index and AAA Diameter Abdominal Aortic Diseases Fabien Lareyre 1,* , Claudine Moratal 2 , Elamine Zereg 3 , Joseph Carboni 4 , Patricia Panaïa-Ferrari 5 , Pascale Bayer 3 , Elixène Jean-Baptiste 1 , Réda Hassen-Khodja 1 , Giulia Chinetti 5 , Juliette Raffort 5 1 Department of Vascular Surgery, University Hospital of Nice, France and Université Côte d’Azur, CHU, Inserm, C3M, Nice, France 2 Université Côte d’Azur, CHU, Inserm, C3M, Nice, France 3 Department of Clinical Biochemistry, University Hospital of Nice, France 4 Department of Vascular Surgery, University Hospital of Nice, France 5 Department of Clinical Biochemistry, University Hospital of Nice, France and Université Côte d’Azur, CHU, Inserm, C3M, Nice, France Introduction: Abdominal aortic aneurysm (AAA) is most often associated with atherosclerosis and corresponding cardiovascular risk factors. However, epidemiological studies have paradoxically highlighted a negative associa- tion between diabetes and AAA. The pathways involved are still poorly understood. Type 2 diabetes is characterized by insulin resistance associated with defects in insulin secre- tion. The aim of this study was to investigate the link be- tween insulin resistance and AAA size. Methods: Patients with non-ruptured AAA were prospec- tively included between January 2016 and February 2017. Patients with AAA caused by specific etiologies such as in- fectious or genetic diseases were excluded. Fasting blood samples were collected to measure glucose, fructosamine, insulin, C-peptide and calculate insulin resistance according to the homeostasis model assessment (HOMA2 IR). The population was divided into 2 groups according to the me- dian value of AAA diameter (< 50 mm et  50 mm). Group differences were compared using non-parametric Mann- Whitney test for continuous variables. Fisher ’ s exact test was used for categorical data. Correlations were determined by non-parametric Spearman’ s correlation coefficient. Results: Fifty-five patients were included, with 87.3% of men. The median age of the cohort was 72 years (66- 79). De- mographic characteristics as well as the presence of cardio- vascular risk factors were similar between patients with AAA 50 mm and those with AAA< 50 mm. Patients with AAA 50 mm had similar fasting glycemia compared to those with AAA< 50 mm (6.1 vs 5.9 mmol/L, p¼0.825). C-peptide and insulin concentrations were significantly higher in patients with AAA 50 mm (0.82 vs 0.54 nmol/L, p¼0.012 and 9 vs 5 mU/L, p¼0.019). This was associated with a higher insulin resistance index (HOMA2 IR index: 1.72 vs 1.26, p¼0.028). Fructosamine concentration was lower in patients with AAA> 50 mm (225.5 vs 251 mmol/L, p¼0.005) and negatively correlated with AAA diameter (r¼-0.55, p<0.0001). Conclusion: Patients with AAA 50 mm had significantly higher insulin resistance index. Further clinical and funda- mental studies are required to determine a causal link be- tween insulin resistance and AAA development. This could, at term, lead to the identification of new therapeutic targets. P-184 Prognostic Role of Ankle-Brachial Index on Cardiac Damage After Carotid Artery Endarterectomy Supra-aortic Arterial & Carotid Diseases George Galyfos 1,2,* , Georgios Sachsamanis 1 , Ioannis Sachmpazidis 1 , Christiana Anastasiadou 1 , Georgios Kastrisios 1 , Sotirios Giannakakis 1 , Anastasios Papapetrou 1 , Gerasimos Papacharalampous 1 , Chrisostomos Maltezos 1 1 Department of Vascular Surgery, KAT General Hospital, Athens, Greece 2 Department of Propedeutic Surgery, University of Athens, Hippocration Hospital, Athens, Greece Introduction: Carotid endarterectomy (CEA) has been associated with an increased risk for postoperative myocardial ischemia. 1 Furthermore, ankle-brachial index (ABI) has been proved to be an independent risk predictor Abstracts e511