Research Article
Serum Interleukin 17 Levels in Patients with Crohn’s Disease:
Real Life Data
Abdurrahman Sahin,
1
Turan Calhan,
2
Mustafa Cengiz,
3
Resul Kahraman,
4
Kubra Aydin,
5
Kamil Ozdil,
6
May Korachi,
5
and H. Mehmet Sokmen
6
1
Department of Gastroenterology, Elazig Education and Research Hospital, Rizaiye Mah. Inonu Caddesi, 23200 Elazig, Turkey
2
Gastroenterology Department, Turkiye Gazetesi Hospital, 34381 Istanbul, Turkey
3
Department of Gastroenterology, Ankara Oncology Education and Research Hospital, 06500 Ankara, Turkey
4
Department of Gastroenterology, Batman State Hospital, 72070 Batman, Turkey
5
Department of Genetics and Bioengineering, Yeditepe University, 34755 Istanbul, Turkey
6
Department of Gastroenterology, Umraniye Training and Research Hospital, 34764 Istanbul, Turkey
Correspondence should be addressed to Abdurrahman Sahin; arahmansmd@yahoo.com
Received 3 June 2014; Accepted 16 June 2014; Published 16 July 2014
Academic Editor: Giuseppe Murdaca
Copyright © 2014 Abdurrahman Sahin et al. Tis is an open access article distributed under the Creative Commons Attribution
License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly
cited.
Te aim of this study was to investigate serum IL17 levels in patients with Crohn’s disease (CD) and to investigate the relationship
between serum IL17 levels with disease activity. Methods. Fify patients with CD and sex- and age-matched 40 healthy controls
were included in the study. Te serum IL17 levels, complete blood count, blood chemistry, erythrocyte sedimentation rate (ESR),
and C-reactive protein (CRP) levels were measured, and Crohn’s disease activity was calculated using Crohn’s disease activity index
(CDAI). Results. Te mean serum IL17 level of CD patients did not difer from those of healthy controls ( > 0.05). Tere was
no diference between the mean serum IL levels of active CD patients and of quiescent CD patients ( > 0.05). However, the
mean IL17 level of active patients was lower than of control subjects ( = 0.02). Serum IL17 was not correlated with infammatory
markers (ESR, CRP, white blood count, platelet count, and albumin) and CDAI. Conclusions. Peripheral blood serum IL17 levels
of CD patients were not higher than of healthy controls, and also, serum IL17 level was not correlated with clinical disease activity.
Peripheral IL17 measurement is not a useful tool for detecting and monitoring Crohn’s disease which is understood to have complex
etiopathogenesis.
1. Introduction
Crohn’s disease (CD) is a chronic relapsing infammatory
disease afecting the gastrointestinal tract and presenting
with extraintestinal manifestations as well. Although the
etiopathogenesis of this disease is not completely understood,
it seems to be infuenced by several environmental factors in
genetically predisposed individuals [1, 2]. T helper lympho-
cytes (T) play an important role in the pathogenesis of CD.
Crohn’s disease is postulated to be an infammatory disease
mediated by T1 cells and recently also by T17 cells [3].
T17 cells are major contributors to several autoimmune
diseases that were previously thought to be T1 cell predom-
inant diseases. Tese autoimmune diseases can be listed as
rheumatoid arthritis, psoriasis, systemic lupus, scleroderma,
multiple sclerosis, infammatory bowel disease, autoimmune
myocarditis, and endometriosis [4]. T17 cells also play an
important role in maintaining intestinal mucosal barrier
function by afecting innate and adaptive responses. Mucosal
T17 cells prevent migration of pathogens from breaking
mucosa to the systemic circulation through the chemotaxis
of neutrophils and macrophages.
One of the important features of T17 cells is to bal-
ance mucosal infammation by regulating the immunogenic
response against self-antigens or intestinal pathogens due
to their relationship with regulatory T cells. Te other is
plasticity, the ability of these cells to diferentiate to other
T cell subgroups under various types of stimulation. T17
Hindawi Publishing Corporation
Disease Markers
Volume 2014, Article ID 690853, 6 pages
http://dx.doi.org/10.1155/2014/690853