Rapid communication R7
Journal of Endocrinology (2000) 167, R7–R10 Accepted 16 October 2000
0022-0795/00/0167-00R7 © 2000 Society for Endocrinology Printed in Great Britain
Online version via http://www.endocrinology.org
The hexapeptide KP-102 (D-Ala-D-
Β-Nal-Ala-Trp-D-Phe-Lys-NH
2
)
stimulates growth hormone release in a cichlid fish (
Oreochromis
mossambicus)
B S Shepherd
1,2
*, S M Eckert
3
, I S Parhar
4
, M M Vijayan
5
,
I Wakabayashi
6
, T Hirano
3
, E G Grau
3
and T T Chen
1
1
Biotechnology Center, University of Connecticut, Storrs, Connecticut 06269, USA
2
T.H. Morgan School of Biological Sciences, 101 Morgan Building, University of Kentucky, Lexington, Kentucky 40506-0225, USA
3
Hawaii Institute of Marine Biology, University of Hawaii, Kaneohe, Hawaii 96744, USA
4
Department of Physiology, Nippon Medical School, Tokyo 113, Japan
5
Department of Biology, University of Waterloo, Ontario N2L 3G1, Canada
6
Department of Medicine, Nippon Medical School, Tokyo 113, Japan
(*To whom correspondence should be addressed, E-mail "bsshep2@pop.uky.edu")
Abstract
Studies in mammals have shown that synthetic Met-
enkephalin derivatives, called growth hormone-releasing
peptides (GHRPs), stimulate growth hormone (GH) release.
The present study was conducted to determine whether the
GHRP, KP-102, specifically stimulates GH release in a teleost.
Tilapia (Oreochromis mossambicus) were given a single
intraperitoneal injection of KP-102 (D-Ala-D-Β-Nal-Ala-Trp-
D-Phe-Lys-NH2) or bovine GHRH1-29-amide or vehicle and
blood was sampled at 1, 6 and 12 h after injection. KP-102 was
administered at two doses of 1 ng/g and 10 ng/g body weight,
whereas GHRH (positive control) was administered at a single
dose of 10 ng/g body weight. Plasma levels of tilapia GH and
prolactins (tPRL177 and tPRL188) were determined by
radioimmunoassay. As expected, GHRH injection
significantly (P <0.001) elevated plasma GH levels (ng/ml) in
tilapia at 6 h post-injection. KP-102 also significantly elevated
GH levels (at the low dose) at 6 (P <0.05) and 12 (P <0.01)
hours post-injection. There were no significant effects on
plasma PRL(s) levels, although mean levels of both PRLs
were elevated at 6 h post-injection. These results show for the
first time that GHRPs stimulate GH release in teleosts and
suggest that the GHRP receptor and possibly a "Ghrelin-like"
ligand are also present in lower vertebrates.
Journal of Endocrinology (2000) 167, R7-R10
Introduction
Growth hormone (GH) and prolactin (PRL) are structurally
related hormones that are secreted by the vertebrate anterior
pituitary, and their secretion is principally under the control of
neuroendocrine factors (e.g., growth hormone-, gonadotropin-
and thyrotropin-releasing hormones and somatostatin and
dopamine). In addition to these regulatory factors, synthetic
Met-enkephalin derivatives (hexapeptides) have also been
shown to stimulate GH, and to some extent, PRL release in
mammals (Bowers et al. 1990, Elias et al. 1995, Bowers
1998). These hexapeptides, also termed growth hormone-
releasing peptides (GHRPs), have been extensively studied
and have been shown to safely stimulate pulsatile GH release
and consequently growth in mammals and humans (Baker
et al. 1984, Thorner et al. 1997, Bowers 1998, Camanni et al.
1998, Mericq et al. 1998).
Studies in mammals have shown that GHRPs stimulate
GH secretion and growth by binding to a novel receptor
(Howard et al. 1996). Recently, a new endogenous protein
termed "Ghrelin", which stimulates GH secretion by binding
to the GHRP receptor, has been reported (Kojima et al. 1999).
These findings argue for the presence of novel neuroendocrine
pathways that control growth-regulating hormones in higher
vertebrates. By contrast, in non-mammalian vertebrates,
GHRP(s) administration, alone, has not been shown to be
active in vivo (Bowers et al. 1984). Information on the activity
of GHRPs in lower vertebrates will provide valuable insight
into the common regulatory mechanisms that control
vertebrate neuroendocrine physiology (Bowers 1998).
Against this background, we examined whether the
GHRP, KP-102, would stimulate GH and/or PRL levels in a
teleost, the tilapia (Oreochromis mossambicus). In addition to
GH, the tilapia pituitary secretes two forms of prolactin
(tPRL177 and tPRL188), one of which contain 177 amino acid
residues, and the other, 188 amino acid residues. These two
prolactins, to which unique functions have been ascribed
(Rubin & Specker 1992, Oshima et al. 1996, Sakamoto et al.
1997, Shepherd et al. 1997), are encoded by separate genes
and share only 69% sequence identity (Specker et al. 1985,
Rentier-Delrue et al. 1989, Yamaguchi et al. 1991). In this
study, we report the effects of KP-102 treatment on circulating
GH and PRL(s) levels in a cichlid teleost.
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