Vol.:(0123456789) 1 3
Molecular Biology Reports
https://doi.org/10.1007/s11033-018-4294-5
ORIGINAL ARTICLE
A natural favonoid lawsonaringenin induces cell cycle arrest
and apoptosis in HT-29 colorectal cancer cells by targeting multiple
signalling pathways
Areeba Anwar
1
· Nizam Uddin
2
· Bina Shaheen Siddiqui
2
· Rafat Ali Siddiqui
3
· Sabira Begum
2
·
Muhammad Iqbal Choudhary
1,2,4
Received: 11 May 2018 / Accepted: 1 August 2018
© Springer Nature B.V. 2018
Abstract
Colorectal cancer is the third most common malignancy in the world having a high mortality rate. Flavonoids possess many
biological activities including anti-cancer activity. lawsonaringenin (LSG) is a favonoid isolated from leaves of Lawsonia
alba Lam. The objective of this study was to demonstrate the anti-cancer potential of LSG in colorectal cancer for the frst
time. The HT-29 cells were treated with LSG or 5-fuoruracil, as a positive control, to determine its efect on cell cytotoxic-
ity by a MTT cell proliferation assay, and cell cycle progression and apoptosis using fowcytometry. We also determined the
mechanisms underlying LSG-mediated growth inhibition of HT-29 cells by by investigating the expression of key oncogenes
and apoptosis genes using q-RT PCR and immunocytochemical analysis. The cell cytotoxicity data showed that the IC
50
value of LSG was signifcantly less than the IC
50
value of 5-FU (50 µM). The anti-proliferative efect of LSG was mediated
by arresting cells in the S phase of the cell cycle which then led to the induction of apoptosis the q-RT PCR and immunocyto-
chemical analysis showed that LSG reduced the expression of ß-catenin (non-phosphorylated) and its downstream signalling
target c-Myc, whereas it increased the phosphorylation of ß-catenin. Furthermore, LSG also downregulated the expression
of oncogene K-Ras and anti-apoptotic proteins, Bcl-2, and Bcl-xL. In conclusion, our data demonstrates that LSG exerted
its anti-tumor activity by arresting the cell cycle in S phase, and by downregulating the expression of oncogenes including
ß-catenin, c-Myc, K-Ras and anti-apoptosis proteins Bcl-2 and Bcl-xL. This study suggests a potential use of natural favo-
noid, lawsonaringenin, to attenuate colorectal cancer growth; however, further pre-clinical/clinical studies are required to
establish its role as a therapeutic agent.
Keywords Colorectal cancer · HT-29 cells · Lawsonia alba Lam. · Apoptosis · ß-Catenin signalling · Flavonoid
Abbreviations
ß-Cat-NP Beta catenin (non-phosphorylated)
ß-Cat-P Beta catenin (phosphorylated)
5-FU 5-Fluorouracil
Bcl-2 B-cell lymphoma 2 protein
Bcl-Xl B-cell lymphoma extra-large protein
Electronic supplementary material The online version of this
article (https://doi.org/10.1007/s11033-018-4294-5) contains
supplementary material, which is available to authorized users.
* Areeba Anwar
areeba_anwar@ymail.com
* Muhammad Iqbal Choudhary
iqbal.choudhary@iccs.edu
1
Dr. Panjwani Center for Molecular Medicine and Drug
Research, International Center for Chemical and Biological
Sciences, University of Karachi, Karachi 75270, Pakistan
2
H.E.J. Research Institute of Chemistry, International Center
for Chemical and Biological Sciences, University of Karachi,
Karachi 75270, Pakistan
3
Nutrition Science and Food Chemistry Laboratory,
Agriculture Research Station, Virginia State University,
Petersburg, VA 23806, USA
4
Department of Biochemistry, Faculty of Sciences, King
Abdulaziz University, Jeddah 21412, Saudi Arabia