Citation: di Polidoro, A.C.; Cafarchio,
A.;Vecchione, D.; Donato, P.; De
Nola, F.; Torino, E. Revealing
Angiopep-2/LRP1 Molecular
Interaction for Optimal Delivery to
Glioblastoma (GBM). Molecules 2022,
27, 6696. https://doi.org/10.3390/
molecules27196696
Academic Editor: Yin Wang
Received: 2 September 2022
Accepted: 4 October 2022
Published: 8 October 2022
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molecules
Article
Revealing Angiopep-2/LRP1 Molecular Interaction for Optimal
Delivery to Glioblastoma (GBM)
Angela Costagliola di Polidoro
1,2
, Andrea Cafarchio
1,3
, Donatella Vecchione
4
, Paola Donato
4
,
Francesco De Nola
4
and Enza Torino
1,2,
*
1
Department of Chemical, Materials and Production Engineering (DICMaPI), University of Naples Federico II,
80125 Naples, Italy
2
Interdisciplinary Research Center on Biomaterials, CRIB, University of Naples Federico II, 80125 Naples, Italy
3
Dipartimento di Medicina e Scienze della Salute Vincenzo Tiberio, University of Molise,
86100 Campobasso, Italy
4
Teoresi Spa—Via F. Imparato, 198 CM2, 80146 Napoli, Italy
* Correspondence: enza.torino@unina.it
Abstract: Background: The family of synthetic peptide angiopeps, and particularly angiopep-2
(ANG-2) demonstrated the ability preclinically and clinically to shuttle active molecules across the
blood–brain barrier (BBB) and selectively toward brain tumor cells. The literature has also proved
that the transport occurs through a specific receptor-mediated transcytosis of the peptide by LRP-1
receptors present both on BBB and tumor cell membranes. However, contradictory results about
exploiting this promising mechanism to engineer complex delivery systems, such as nanoparticles, are
being obtained. Methodology: For this reason, we applied a molecular docking (MD)-based strategy
to investigate the molecular interaction of ANG-2 and the LRP-1 ligand-binding moieties (CR56 and
CR17), clarifying the impact of peptide conjugation on its transport mechanism. Results: MD results
proved that ANG-2/LRP-1 binding involves the majority of ANG-2 residues, is characterized by
high binding energies, and that it is site-specific for CR56 where the binding to 929ASP recalls a
transcytosis mechanism, resembling the binding of the receptor to the receptor-associated protein.
On the other hand, ANG-2 binding to CR17 is less site-specific but, as proved for apolipoprotein
internalization in physiological conditions, it involves the ANG-2 lysin residue. Conclusions: Overall,
our results proved that ANG-2 energetic interaction with the LRP-1 receptor is not hindered if specific
residues of the peptide are chemically crosslinked to simple or complex engineered delivery systems.
Keywords: angiopep-2; glioblastoma; optimal design; molecular docking
1. Introduction
Effective delivery to the central nervous system (CNS) is an incredibly challenging
task that is still mostly unaccomplished despite the numerous efforts outlined in the recent
literature on this topic. Invasive and non-invasive approaches, both relying on externally
applied stimuli or physiological mechanisms, as well as different routes of administration
have been extensively explored to improve active agent penetration into the CNS [1,2].
The angiopep family of peptides has been derived from the Kunitz domain of human
aprotinin [3]. These peptides are able to cross the blood–brain barrier (BBB) and have been
used to facilitate the delivery of pharmacological agents to the brain, for example to target
glioblastoma tumors and recurrent brain metastases of pre-treated breast cancers [4]. In
particular, angiopep-2 (ANG-2) has higher transcytosis capacity and higher brain volume
of distribution than aprotinin. Like aprotinin, angiopep-2 interacts with low-density
lipoprotein receptor-related protein 1 (LRP1) which is thought to promote its delivery
across the BBB via receptor-mediated transcytosis (RMT) [5]. The LRP-1 receptor is a
600 kDa member of the low-density lipoprotein receptor family physiologically involved in
the transcytosis of many proteins and peptides across the BBB [6].
Molecules 2022, 27, 6696. https://doi.org/10.3390/molecules27196696 https://www.mdpi.com/journal/molecules