Volume 4 • Issue 5 • 1000160
J Drug Metab Toxicol
ISSN: 2157-7609 JDMT, an open access journal
Research Article Open Access
Akın et al., J Drug Metab Toxicol 2013, 4:5
DOI: 10.4172/2157-7609.1000160
Research Article Open Access
*Corresponding authors: Şakir Özgür Keşkek, Ministry of Health, Adana Numune
Education and Training Hospital, Internal Medicine Clinic, Yüreğir, Adana, Turkey,
Tel: +905052996942; E-mail: drkeskek@yahoo.com
Received November 13, 2013; Accepted December 18, 2013; Published
December 20, 2013
Citation: Akın A, Keşkek ŞÖ, Kılıç DA, Aliustaoğlu M, Keşkek NŞ (2013) The
Effects of N-acetylcysteine in Patients with Amanita phalloides Intoxication. J Drug
Metab Toxicol 4: 160. doi:10.4172/2157-7609.1000160
Copyright: © 2013 Akın A, et al. This is an open-access article distributed under
the terms of the Creative Commons Attribution License, which permits unrestricted
use, distribution, and reproduction in any medium, provided the original author and
source are credited.
Abstract
Objectives: Mushroom intoxication with Amanita phalloides has a high incidence throughout the world.
Treatment for this intoxication is similar in different centers, but N-acetylcysteine is rarely used. In this study, we
aimed to investigate the effects of N-acetylcysteine treatment in patients with Amanita phalloides intoxication.
Methods: A total of 40 patients with Amanita phalloides intoxication were included in this retrospective study. The
study group consisted of 24 patients who were administered N-acetylcysteine in addition to the standard regimen;
the control group consisted of 16 patients who were treated only with the standard treatment. Treatment results and
biochemical measurements of groups were compared.
Results: According to the biochemical measurements, it was found that patients in the control group were
affected more seriously by Amanita phalloides than those in the study group. The mortality rate was lower in the
study group (4.4% vs. 18.7% in the control group).
Conclusions: Amanita phalloides intoxication can be successfully treated with N-acetylcysteine in addition to
the standard regimen. Signifcantly, the simplicity of administration, good tolerance, and an affordable cost make
N-acetylcysteine a viable option for the treatment of Amanita phalloides intoxication. The low mortality rate presented
in the study group may be ascribed to N-acetylcysteine administration.
The Effects of N-acetylcysteine in Patients with Amanita phalloides
Intoxication
Ahmet Akın
1
, Şakir Özgür Keşkek
2
*, Didem Aydın Kılıç
1
, Mehmet Aliustaoğlu
1
and Nedime Şahinoğlu Keşkek
3
1
Ministry of Health, Dr Lütf Kırdar Kartal Education and Training Hospital, Internal Medicine Clinic, Istanbul, Turkey
2
Ministry of Health, Adana Numune Education and Training Hospital, Internal Medicine Clinic, Yüreğir, Adana, Turkey
3
Ministry of Health, Adana Numune Education and Training Hospital, Ophthalmology Clinic, Yüreğir, Adana, Turkey
Keywords: Amanita phalloides; Intoxication; N-acetylcysteine
Introduction
Mushroom intoxication is the most common vegetal intoxications
in Turkey and in other parts of the world [1,2]. It may result in several
disorders, ranging from mild gastroenteritis to severe fulminant hepatic
failure. Te main cause of mortality is amatoxin, Amanita phalloides (A.
phalloides) toxin, which leads to very high mortality rates up to >20% in
adults and >50% in children [3]. Amatoxin noncompetitively inhibits
RNA polymerase II or B, which is dependent on DNA. Bromelain,
cysteine proteinase, cathepsin, cathepsin L, and papain are released
[4]. Inhibition of mRNA synthesis in hepatocytes causes a decrease
in coagulation factor and immunoglobulin production. In addition,
another toxin, phallotoxin, tends to adhere to microflament structures
and causes cholestasis by stabilizing F actin flaments. Although the
main target tissue of A. phalloides is the liver, kidney cells may also be
afected. Expectedly, intoxication frequency exhibits seasonal variation,
with a signifcant increase during fall, concurrent with the rains.
N-acetylcysteine (NAC) has anti-infammatory, antioxidant,
inotropic, and vasodilating efects that improve microcirculatory blood
fow and oxygen delivery to vital organs [5]. It also supplies sulfydryl
groups to act as a substrate for detoxifying reactive toxic intermediates
[6]. NAC may beneft patients with non-acetaminophen-induced acute
liver failure by improving systemic hemodynamics via tissue oxygen
delivery or other favorable efects on the acutely injured liver. Benefts
are seen when NAC is used in the early stages of liver failure [7]. Studies
on the efects of NAC on Amanita phalloides intoxication are very rare.
Furthermore, in some previous research, NAC was administered with
the standard regimen, which includes silibinin and other treatments
such as plasmapheresis, so it is difcult to show the clear efect of NAC
on A. phalloides intoxication in these studies [8-10]. In the present
research, we aimed to investigate the benefts of N-acetylcysteine
treatment in addition to the standard treatment in patients with A.
phalloides intoxication.
Material and Methods
In this study, data from 40 patients with A. phalloides intoxication
who were treated at the Internal Medicine Clinic of our institute from
2001 to 2006 were studied retrospectively. Te institutional review
board of our hospital approved the study. Mushroom intoxication
patients with any comorbidity and/or patients who were admitted to
the hospital more than 24 hours afer ingestion were excluded.
Patients were divided into two groups. Te study group consisted
of 24 patients who were administered N-acetylcysteine in addition
to the standard regimen, while the control group consisted of
16 patients who were treated only with the standard treatment.
According to the standard treatment, nasogastric tubes and gastric
enemas were applied to patients who presented in the frst 24 hours.
Additionally, hemoperfusion was performed, and intravenous
penicillin G (1 million units/kg/day) infusion, activated charcoal (5
g/day), and lactulose (4 g/day) were administered in three divided
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ISSN: 2157-7609
Journal of Drug Metabolism and
Toxicology