REV.CHIM.(Bucharest) ♦ 70 ♦ no 12 ♦ 2019 4400 http://www.revistadechimie.ro Dental Dysplasia as Structural Anomalies in Child Oral Pathogeny MADALINA MATEI 1 , ODETTE-ELENA LUCA 2 *, MIHAELA BLAJ 2 *, ANAMARIA ZAHARESCU 1 , CATALIN PLESEA CONDRATOVICI 1 , DANIELA ANISTOROAEI 2 , CRISTIAN ROMANEC 2 Dunarea de Jos University of Galati, Faculty of Medicine and Pharmacy, 47 Domneasca Str., 800008, Galati, Romania Grigore T. Popa, University of Medicine and Pharmacy, Faculty of Dental Medicine, 16 Universitatii Str., 700115, Iasi, Romania Dental dysplasia such as structural anomalies, complex pathogenic foundations, although of a relatively low incidence in the child oral pathology is of a a wide interest, attested to a considerable wealth of scientific material reserved in the specialized literature. The prevalence of defective enamel on permanent teeth in the maxilla is smaller in the jaw. The permanent superior incisors are in the first place from the point of view of frequency in the enamel defects, usually symmetric, either at the 2 central, or at all the 4. The first permanent molars and the premolars are permanently affected to a lesser extent, and on canines injuries occur very rarely, only in very serious cases (genetic). We have examined a number of 300 children school communities, with the chronological age between 6 and 11 years 195 girls (65%) and 105 boys (35%). On distribution of children in age groups, we found a concentration of distribution at the age of 8-9 yearsChanges to the DDE index in order to be efficiently and flawlessly divided by enamel defects, being suitable for screenings, but also for more detailed and larger studies, when they can be adapted to the research requirements. The most frequent affectation of the upper permanent incisors correlated with permanent primary molars, implies an intensification of the dental education activity oriented to the young schoolchildren, then the awareness of the dental factor in the facial aesthetics appears late, during puberty-adolescence when the lesions remain aggravate and the oral rehabilitation is complex and difficult, in these cases. Keywords: dental dysplasia, progress defects, enamel development. The interest for the development of the possibilities and methods of clinical and paraclinical evaluation of dental dysplasia appears in the literary version from 1916, when MC Key describes a discromy of the enamel called the " motled" enamel (spotted) that favored in establishing that the fluoride in the water is one of the causes of fluorosis and that it is an association regarding the creation of the fluor concentration in water and the discharge of caries in the studied population [1-3]. Embryogenesis as an oral cavity including through the dental system is closely related to extreme cephalic ontogenesis. The ontogenetic dynamics of the cells, in the formation of the human embryo is variable as the growth rate in the embryos is not the same, with large differences in terms of growth rate. Growth is a process of exchange which changed the zygote into a multicellular organism. The cell has put the codes of care of the genetics, by expressing them, it contributes to the formation of the maxillary bones and teeth [4-6]. In the 13 mm embryo, the epithelium as a primitive oral cavity is bistratified. It has a layer of high, prismatic basal cells and a superficial layer in superficial format up to a single row of cells is applied with its help in much glycogen cytoplasm. At the end of the first embryonic month, the cells in their care will be dental organs: ectodermal, ectomezodermal, ectodermal. Enamel formation (amelogenesis) is the result of cell secretion processes and there are three phases: the precursors, the cells differentiated by chronological age, are aligned so that the cells that are older and the youngest cervical; secretory, when there are strings of ameloblasts withdraw from the dentin in full, but no extension is left inside the matrix, which can be degraded, being replaced by water; maturation starts at this time, when the full thickness of an enamel format is increased, protein and water are eliminated and protruded the ions. Temporary teeth undergo a much more accelerated maturation process. When determining the enamel, the differences regarding the orientation of the enamel prisms, which, at the level of the cracks and the occlusal holes are almost parallel with the longitudinal axis of the temporal teeth [7-9]. *email: lucaodette@yahoo.com; miblaj@yahoo.com